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Price Tab-Cap 3 G 2.55 0.0026 TABLETS 2.65 0.0027 TABLETS 2.70 0.0027 TABLETS 3.05 0.0030 TABLETS 3.12 0.0031 TABLETS 3.58 0.0037 TABLETS 3.64 0.0037 TABLETS 3.91 0.0039 TABLETS 4.10 0.0041 TABLETS 9.29 0.0093 TABLETS Supplier Median Price Tab-Cap 0.0034 High Low Ratio 3.58 2.78 0.0028 TABLETS 3.03 0.0030 TABLETS 0.00 0.0037 TABLET 20.10 0.0040 TABLETS 4.45 0.0045 TABLETS. The UAB constructed a web-based survey tool for this program. Physicians who accessed the UAB Continuing Medical Education CME ; site participated in this CME-accredited study 1 CEU ; from January 16, 2004 through April 30, 2004. Participants answered questions characterizing their demographics and reading habits, and rated 7 factors that may influence their interpretation of the medical literature 1 least important to 5 most important ; . The physicians were randomly assigned to read one of the two articles JAMA, n 73; IJCP, n 92 ; , provided as an Adobe Acrobat PDF, as part of the CME program. A third group of physicians n 134 ; did not read any articles and was used as a control for a case study assessment of the influence of the test articles on treatment outcomes analysis underway ; . Those assigned an article ranked its influence on their approach to, and understanding of, the prevention and management of osteoporosis 1 no impact to 10 definitely impacts ; based on the science, quality of data, presentation, and other factors they felt influenced their perception of the article. In addition, they rated the article according to the READER Relevance, Education, Applicability, Discrimination, overall Evaluation ; literature rating system for clinicians.5-6 Six messages for each article previously identified by the AMMS of which 3 were scored as the key clinical concepts of the article, and 3 were considered to be related to the study ; were provided to the UAB. Readers in the JAMA group were asked to identify the 3 key concepts for the article from the list provided. The study was powered to detect a difference between groups at the P 0.05 level. Statistical analyses were conducted by the UAB, because theophylline sodium. The bulk of medical men of science believe that the use of antibiotics in the treatment of strep throat should be avoided unless absolutely necessary. Because theophylline clearance is variable, and maintaining an appropriate serum level is important in preventing side effects 79 ; , monitoring of theophylline levels is recommended 48.
Author keywords: caffeine; theophylline; metal cation; binding constant; ftir; uv– visible spectroscopy ftir, fourier transform infrared corresponding author. DOSAGE AND ADMINISTRATION, CONTRAINDICATIONS, and WARNINGS ; . Alternatively, treatment can begin within 3 to 10 days of the acute event see DOSAGE AND ADMINISTRATION ; . CONTRAINDICATIONS Myocardial Infarction Metoprolol tartrate is contraindicated in patients with a heart rate 45 beats min; second- and third-degree heart block; significant first-degree heart block P-R interval 0.24 sec systolic blood pressure 100 mmHg; or moderate-to-severe cardiac failure see WARNINGS ; . WARNINGS Myocardial Infarction Cardiac Failure: Sympathetic stimulation is a vital component supporting circulatory function, and beta blockade carries the potential hazard of depressing myocardial contractility and precipitating or exacerbating minimal cardiac failure. During treatment with metoprolol tartrate, the hemodynamic status of the patient should be carefully monitored. If heart failure occurs or persists despite appropriate treatment, metoprolol tartrate should be discontinued. Bradycardia: Metoprolol tartrate produces a decrease in sinus heart rate in most patients; this decrease is greatest among patients with high initial heart rates and least among patients with low initial heart rates. Acute myocardial infarction particularly inferior infarction ; may in itself produce significant lowering of the sinus rate. If the sinus rate decreases to 40 beats min, particularly if associated with evidence of lowered cardiac output, atropine 0.25 to 0.5 mg ; should be administered intravenously. If treatment with atropine is not successful, metoprolol tartrate should be discontinued, and cautious administration of isoproterenol or installation of a cardiac pacemaker should be considered. AV Block: Metoprolol tartrate slows AV conduction and may produce significant first- P-R interval 0.26 sec ; , second-, or third-degree heart block. Acute myocardial infarction also produces heart block. If heart block occurs, metoprolol tartrate should be discontinued and atropine 0.25 to 0.5 mg ; should be administered intravenously. If treatment with atropine is not successful, cautious administration of isoproterenol or installation of a cardiac pacemaker should be considered. Hypotension: If hypotension systolic blood pressure 90 mmHg ; occurs, metoprolol tartrate should be discontinued, and the hemodynamic status of the patient and the extent of myocardial damage carefully assessed. Invasive monitoring of central venous, pulmonary capillary wedge, and arterial pressures may be required. Appropriate therapy with fluids, positive inotropic agents, balloon counterpulsation, or other treatment modalities should be instituted. If hypotension is associated with sinus bradycardia or AV block, treatment should be directed at reversing these see above ; . Bronchospastic Diseases: PATIENTS WITH BRONCHOSPASTIC DISEASES SHOULD, IN GENERAL, NOT RECEIVE BETA BLOCKERS. Because of its relative beta1 selectivity, metoprolol tartrate may be used with extreme caution in patients with bronchospastic disease. Because it is unknown to what extent beta2-stimulating agents may exacerbate myocardial ischemia and the extent of infarction, these agents should not be used prophylactically. If bronchospasm not related to congestive heart failure occurs, metoprolol tartrate should be discontinued. A theophylline derivative or a beta2 agonist may be administered cautiously, depending on the clinical condition of the patient. Both theophylline derivatives and beta2 agonists may produce serious cardiac arrhythmias. PRECAUTIONS General Metoprolol tartrate should be used with caution in patients with impaired hepatic function. Drug Interactions and albenza.

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REFERENCES 1. Ring IT, Firman D. Reducing indigenous mortality in Australia: lessons from other countries. Med J Aust 1998; 169: 528-33. Commonwealth Grants Commission. Report on indigenous funding 2001. Canberra: CGC; 2001. : cgc.gov.au IFI Downloads Final Report IFIReport [Accessed September 2003] 3. Paradies Y, Cunningham J. Placing Aboriginal and Torres Strait Islander mortality in an international context. Aust N Z J Public Health 2002; 26: 11-6. Couzos S, Murray R. Aboriginal primary health care. An evidence-based approach. 2nd ed. Melbourne: Oxford University Press; 2003. 5. Keys Young. Market research into Aboriginal and Torres Strait Islander access to Medicare and the Pharmaceutical Benefits Scheme. Canberra: Health Insurance Commission; 1997. 6. Australian Institute of Health and Welfare. Expenditures on health services for Aboriginal and Torres Strait Islander people, 1998-99. Canberra: Australian Institute of Health and Welfare and Commonwealth Department of Health and Aged Care; 2001. 7. National Medicines Policy 2000. Canberra: Commonwealth Department of Health and Aged Care; 1999. : health.gov.au haf nmp pdf nmp2000 [Accessed September 2003] 8. Central Australian Rural Practitioners Association. Standard treatment manual for health workers. 4th ed. Alice Springs: CARPA; 2003. TRINORDIOL ethinylestradiol 30 micrograms, levonorgestrel 50 micrograms; ethinylestradiol 40 micrograms, levonorgestrel 75 micrograms; TRIPTORELIN Decapeptyl SR ; m r injection 42mg, 15mg TRIPTORELIN m r injection 15mg TRIZIVIR abacavir 300mg, lamivudine 150mg, zidovudine 300mg ; tablets TROPICAMIDE single use eye drops 1% TROSPIUM tablets 20mg TUBERCULIN PPD RT 23 SSI 2 T.U. 01ml and 10 T.U. 01ml for Mantoux Test UNIPHYLLIN CONTINUS The9phylline ; m r tablets 200mg, 300mg, 400mg URSODEOXYCHOLIC ACID capsules 250mg UVISTAT ULTRABLOCK OTC cream SPF 30 VALPROIC ACID e c tablets 250mg, 500mg VALSARTAN capsules 40mg, 80mg, 160mg VANCOMYCIN capsules 125mg; infusion 500mg, 1 gram VARICELLA-ZOSTER LIVE vaccine VECURONIUM injection 10mg VENLAFAXINE tablets 375mg, 75mg; m r capsules 75mg, 150mg VERAPAMIL tablets 40mg, 80mg, 120mg; m r tablets 120mg Half Securon SR ; , 240mg Securon SR oral solution 40mg 5ml; injection 5mg 2ml VIGABATRIN tablets 500mg; sachets 500mg VINBLASTINE injection 10mg VINCRISTINE injection 1mg 1ml VINDESINE injection 5mg VINORELBINE capsules 20mg, 30mg; injection 10mg 1ml VITAPRO VITLIPID N infant injection VOLUMATIC VOLUVEN infusion WARFARIN tablets 500 micrograms, 1mg, 3mg, 5mg WATER FOR INJECTION WATER FOR IRRIGATION XALACOM latanaprost 50 micrograms, timolol 5mg ml ; eye drops XYLOMETAZOLINE OTC paediatric nasal drops 005%; nasal spray 01% XYLOPROCT ointment includes hydrocortisone acetate 0275%, lidocaine 5% ; YELLOW SOFT PARAFFIN OTC ZIDOVUDINE infusion 200mg 20ml requires dilution capsules 250mg; syrup 50mg 5ml and albendazole. Is there evidence that low background levels of theophylline are synergistic.

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Although the adverse effects of theophylline are related to the plasma concentration, the drug tends to be less well tolerated, even at low doses, than other second line controller drugs such as leukotriene antagonists or long acting. The mortality from theophylline related seizures approaches 30 per cent and glimepiride.

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Activity theory a good synthesis of its theoric basis and its applications for design can be found in Nardi [1996] which stems from culturalhistorical psychology [Vygotskij 1978] can provide representations of what are we saying with its concepts of subject who performs an action ; tool the instrument which mediates the action ; object the goal of the subject ; . A graphic representation of the three situations is given in Figure 2. It is clear that physician, patient and pharmacist are driven in their activities by different objects, i.e. they have different motives for using the same tool. But there is not a balanced relationship between the three subjects: in fact it is the physician who creates the tool "discharge document" using another tool, ARGOS software. We can say that the tool "discharge document" has not only a different use but also a different value for the three subjects: from what we have observed physicians often consider discharge activity a sort of sheer burocratic work, consisting of the boring filling in of documents, that steals time from the "real" clinic work. On the other hand, patients highly depend on this paper for an activity that many times is managed alone: self-medication at home. Pharmacists are rather interested in having a clear comprehension of the document in order to manage a safe dispensing. The object "fast and global understanding" is a temporally delayed goal: it will serve only in case of the patient going back into hospital. The object "full understanding ." is in fact achieved by the primary users of the tool, i.e. patients, rather than the physicians. In other words we have the patients that cannot decide how to arrange information which is vital to their wellbeing; the tool that holds such information is instead created by subjects that do not have the same goal, i.e. the physicians. This situation leads to the production of a document which does not fully support patients and pharmacists in achieving their goals breakdown. Management Step 3b Non-Responder patient who fails to respond adequately to Step 1 ; : Intensify treatment as follows: Salbutamol beta2 agonist ; as above, or, if no improvement after 30-minutes of continuous inhalation: Salbutamol, IV, loading dose 0.5 micrograms kg, followed by 0.2 micrograms kg minute. May be increased by 0.1 micrograms kg every 15 minutes to a maximum of 4 micrograms kg minute. PLUS Ipratropium bromide, nebulised, 1 mL in 1 0.9% sodium chloride solution per dose 4 hourly or 0.1 mL kg 4 hourly PLUS Prednisone, oral, 2 mg kg 24 hours as a single daily dose for 7 days. Taper dosage to stop over 7 days. If oral steroids are not available, use: Dexamethasone, IV, 0.4 mg kg one dose ; . Not to be repeated. Assess response to intensified treatment. If non-responsive, admit to intensive care unit for further management by a specialist paediatrician. Patients not currently receiving theophylline preparations: loading dose, slow IV, 46 mg kg over 30 minutes diluted in 5% dextrose water or 0.9% sodium chloride solution maintenance dose, IV, 6 months9 years, 0.51 mg kg hour, 9 years16 years, 0.50.8 mg kg hour. Patients currently on theophylline preparations with no signs of theophylline toxicity: loading dose, slow IV, 1.52.5 mg kg over 30 minutes diluted in 5% dextrose water or 0.9% sodium chloride solution maintenance dose, IV, 6 months9 years, 0.51 mg kg hour, 9 years16 years, 0.50.8 mg kg hour. Precautions for use of aminophylline: - Monitor blood levels. - Only to be used in an intensive care setting, under supervision of a specialist paediatrician, if there is no response to other treatment modalities. - Do not use if IV salbutamol was used. - Rectal use is contraindicated. Maintain hydration. Comments and anacin.
Figure 14: comparison of model simulation and experimental release profiles of theophylline from ca 398-10 nf.

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Y s e DTP #1. Is the patient taking theophylline on a daily basis as prescribed? T d y Date o a' s and panadol.

2003 STA HealthCare Communications Inc. All rights reserved. Hypertension Canada is published four times a year, in an English and a French edition, by STA HealthCare Communications Inc, 955 Boul. St. Jean, Suite 306, Pointe-Claire, Quebec H9R 5K3. Subscriptions are available upon request to anyone interested in hypertension. All articles published in Hypertension Canada are the sole responsibility of the authors, and do not necessarily reflect the opinions of the Canadian Hypertension Society or STA HealthCare Communications Inc. Legal delivery first quarter 2003. Articles may be submitted, in either English or French, to the Editorial Office, Hypertension Canada, The Toronto Western Hospital, University Health Network, 399 Bathurst St, Toronto, Ontario, M5T 2S6. FAX: 416 ; 603-7919. Deadline for submissions for the next issue is April 15, 2003. Canadian Publications Mail Sales Product Agreement #40063348, for example, half life of theophylline. Tell your health care provider if you are taking any other medicines, especially any of the following: anticoagulants eg, warfarin ; , aspirin, heparin, or nonsteroidal anti-inflammatory drugs nsaids ; eg, ibuprofen ; because the risk of bleeding may be increased antacids because the effectiveness of ticlid may be decreased carbamazepine or theoph6lline because the actions and side effects may be decreased by ticlid cyclosporine or phenytoin because the effectiveness may be decreased by ticlid this may not be a complete list of all interactions that may occur and acetaminophen.

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Epithelial hyporesponsiveness after colitis: role of inducible nitric oxide synthase. J Physiol 1999, 276: G703G710 Hubel KA, Renquist KS: Ion transport in normal and inflamed human jejunum in vitro: changes with electrical field stimulation and theophylline. Dig Dis Sci 1990, 35: 815 Crowe SE, Luthra GK, Perdue MH: Mast cell mediated ion transport in the intestine from patients with and without inflammatory bowel disease. Gut 1997, 41: 785792 Murthy SNS, Cooper HS, Shim H, Shah RS, Ibrahim SA, Sedergran DJ: Treatment of dextran sulfate sodium-induced murine colitis by intracolonic cyclosporin. Dig Dis Sci 1993, 38: 17221734 Egger B, Procaccino F, Sarosi I, Tolmas J, Buchler MW, Eysselein VE: Keratinocyte growth factor ameliorates dextran sodium sulfate colitis in mice. Dig Dis Sci 1999, 44: 836 Dieleman LA, Ridwan BU, Tennyson GS, Beagley KW, Bucy RP, Elson CO: Dextran sulfate sodium-induced colitis occurs in severe combined immunodeficient mice. Gastroenterology 1994, 107: 1643 Axelsson L-G, Lanndstrom E, Goldschmidt TJ, Gronberg A, BylundFellenius A-C: Dextran sulfate sodium DSS ; induced experimental colitis in immunodeficient mice: effects in CD4 -cell depleted, athymic and NK-cell depleted SCID mice. Inflamm Res 1996, 45: 181191 Shintani N, Nakajima T, Sugiura M, Murakami K, Nakamura N, Kagitani Y, Mayumi T: Proliferative effect of dextran sulfate sodium DSS ; pulsed macrophages on T cells from mice with DSS-induced colitis and inhibition of effect by IgG. Scand J Immunol 1997, 46: 581586 Hirono I, Kuhara K, Hosaka S, Tomizawa S, Golberg L: Induction of. Consequently, it is in the short-term drug that helps you to take it more often than prescribed and follow your doctor's instructions especially about the dangers of kids abusing prescription medicines has run on television and in addition, are less expensive than most prescription sedatives currently available and anafranil.
Non-nicotine aid to smoking cessation Zyban bupropion hydrochloride ; is the first smoking cessation agent available in tablet form. It is unclear how bupropion, an antidepressant, works. It has been shown to affect noradrenergic and or dopaminergic mechanisms in the brain which have been implicated as pathways of nicotine addiction. Its efficacy in smoking cessation was demonstrated in clinical trials where it was used with smoking cessation counselling: the 12-month abstinence rates achieved with Zyban were almost twice those obtained with placebo and in a specific study, approximately twice the rate of a nicotine patch. Side effects: The most common are dry mouth, insomnia and dizziness. Its use is also associated with a dose-dependent risk of seizure thus higher than recommended doses should not be prescribed. Contraindications: Patients treated with Wellbutrin SR the antidepressant ; or any other medications that contain bupropion, patients with a seizure disorder, patients with a current or prior diagnosis of bulimia or anorexia nervosa, patients currently or recently treated with a monoamine oxidase MAO ; inhibitor, patients who have shown an allergic reaction to bupropion or the other ingredients that make up Zyban. Caution: It should be administered with extreme caution in patients with a history of seizure, cranial trauma, or other predisposition s ; or higher risk toward seizure, or patients treated with other agents e.g., antipsychotics, antidepressants, theophylline, systemic steroids, etc. ; or treatment regimens e.g., abrupt discontinuation of benzodiazepine ; that lower seizure threshold. Please note that antidepressants can precipitate manic episodes in bipolar disorder patients and may activate latent psychosis in other susceptible individuals. Zyban is not recommended during pregnancy or nursing and its safety in pediatric patients is unknown.

This drug should be taken one week before entering a malaria area, weekly while there and weekly for 4 weeks after leaving the malaria area and clomipramine and theophylline, for instance, theophyllinw drugs. Drug Interactions. Ciprofloxacin may increase levels of theophyllin4 and caffeine, other vitamin and drug products may reduce availability of ciprofloxacin. Hypersensitivity. Pseudomembraneous colitis. Tendonitis tendon rupture. Photosensitivity. Use of an antibiotic inappropriately for example when exposure has not been confirmed ; can lead to the emergence of resistant strains of bacteria. The usefulness of ciprofloxacin hydrochloride as an antibiotic may be lost if widespread use occurs. The safety and effectiveness of ciprofloxacin in patients less than 18 years of age is not established except for post-exposure use in inhalational anthrax. Ciprofloxacin causes arthropathy in juvenile animals. Fluroquinolones are not generally recommended during pregnancy because of their known association with arthropathy in adolescent animals. However, animal studies have not shown evidence of teratogenicity related to exposure to ciprofloxacin 2 ; . A 1999 consensus statement by the Working Group on Civilian Biodefense recommends that ciprofloxacin be used at usual adult doses in pregnant women for therapy and postexposure prophylaxis following anthrax exposure 3. The uk commitee on safety of medicines ukcsm ; advises that hypericum should not be used with indianvir, warfarin, cyclosporin, oral contraceptives, digoxin and theophylline and aralen.

Table 6.7. Medical conditions occurring more frequently in patients with bipolar disorder compared to the general population Type 2 diabetes Metabolic syndrome Cardiovascular disease Obesity Chronic obstructive pulmonary disease Migraine Hepatitis C HIV Lower back pain Asthma Allergies Dementia. A.G. Halpert et al. Neuroscience and Biobehavioral Reviews 26 2002 ; 6167 [24] Kalivas PW. Histamine-induced arousal in the conscious and pentobarbital-pretreated rat. J Pharmacol Exp Therap 1982; 222: 3742. [25] Koob GF. Neural mechanisms of drug reinforcement. Annals NY Acad Sci 1992; 654: 17191. [26] Malcolm R, Miller WC. Dimenhydrinate Dramamine ; abuse: hallucinogenic experiences with a proprietary antihistamine. J Psychiat 1972; 128: 10123. [27] Manning C, Scandale L, Manning EJ, Gengo FM. Central nervous system effects of meclizine and dimenhydrinate: evidence of acute tolerance to antihistamines. J Clin Pharmacol 1992; 32: 9961002. [28] McKearney JW. Stimulant actions of histamine H1 antagonists of operant behaviour in the squirrel monkey. Psychopharmacology 1982; 77: 1568. [29] McKearney JW. Relative potencies of histamine H1 antagonists as behavioural stimulants in the squirrel monkey. Psychopharmacology 1985; 86: 3801. [30] McKim WA. The effect of cafene, theophylline and amphetamine on operant responding of the mouse. Psychopharmacology 1980; 68: 1358. [31] Mumford GK, Holtzman SG. Methylxanthines elevate reinforcement threshold for electrical brain stimulation: role of adenosine receptors and phosphodiesterase inhibition. Brain Res 1990; 528: 328. [32] Mumford GK, Silverman K, Grifths RR. Reinforcing, subjective, and performance effects of lorazepam and diphenhydramine in humans. Exp Clin Psychopharmacol 1996; 4: 42130. [33] Muth ER, Jokerst M, Stern RM, Koch KL. Effects of dimenhydrinate on gastric tachyarrhythmia and symptoms of vection-induced motion sickness. Aviat Space Environ Med 1995; 66: 10415. [34] Niemegeers CJE, Awouters FHL, Janssen PAJ. The in vivo pharmacological prole of histamine H1 ; antagonists in the rat. Drug Dev Res 1982; 2: 55966. [35] Oliver M, Stenn PG. Is there a risk for dependency with therapeutic doses of dimenhydrinate? Psychosomatics 1993; 34: 459. [36] Persson CGA. Overview of the effects of theophylline. J Aller Clin Immunol 1986; 78: 7807. [37] Preston KL, Wolf B, Guarino JJ, Grifths RR. Subjective and behavioural effects of diphenhydramine, lorazepam and methocarbamol: evaluation of abuse liability. J Pharmacol Exp Therap 1992; 262: 70720. [38] Privou C, Knoche A, Hasenohrl RU, Huston JP. The H1- and H2histamine blockers applied to the nucleus basalis magnocellularis region modulate anxiety and reinforcement related processes. Neuropharmacology 1998; 37: 101932. [39] Richelson E. Tricyclic antidepressants: interactions with histamine and muscarinic acetylcholine receptors. In: Enna SJ, editor. Antidepressants: neurochemical, behavioural, and clinical perspectives, New York: Raven Press, 1981. p. 5373. [40] Robinson TE, Berridge KC. The neural basis of drug craving: an incentive-sensitization theory of addiction. Brain Res Rev 1993; 18: 24791. [41] Rowe C, Verjee Z, Koren G. Adolescent dimenhydrinate abuse: resurgence of an old problem. J Adolesc Heal 1997; 21: 479. World Health Organization. Tuberculosis and gender. Website: : who.int tb dots gender en. Consequently, ophthalmologists, out-patient surgical facilities, hospitals and other health care providers may be reluctant to purchase our products if they do not receive substantial reimbursement for the cost of our products and for procedures performed using our surgical medical device products from third-party payors, including medicare and medicaid in the united states and health insurance programs, both governmental and private, because theophylline 400.
Oxytocin . 115 Paclitaxel Taxol ; . 116-117 Papaverine HCL . 118 Paracetamol . 119 Paraquat . 120 Paraxanthine . 45, 140 Peptides 121 Perylene . 122-123 Phencyclidine PCP ; . 124 Phenobarbital . 28, 30, 33-34 Phensuximide . 153 2-Phenyl-2ethylmalonamide Phenytoin . Phthalic Acid . 146 p-Hydroxyphenobarbital Picloram . Prednisolone . 149-152 Prednisolone Acetate . 149 Prednisone . 126 Primidone . 28, 30 Procainamide . 127-129 Prochlorperazine Edisylate . 130 Progesterone . 151-152 Promethazine HCL . 133 Propiophenone . 147-148 Propranolol . 134-135, 160 Propylparaben . 37-38 Protriptyline HCL . 173 Pseudoephedrine HCL . 136-139 Pyridoxal . 180 Pyridoxamine Dihydrochloride . 180 Pyrodoxine HCL . 180 Quercetin . Quinidine sulfate . 141 Ranitidine . 63-64, 142-144 Riboflavin . 180-181 Salbutamol . 18-19 Salicylic acid . 145-146 Secobarbital . 33-34 Simazine . 172 Succinimide . 28, 153 Succinylsulfathiazole . 96, 154 Sulfadiazine . 97, 154-157 Sulfadimethoxine . 156-157 Sulfadimidine . 156-157 Sulfamerazine . 97, 154-157, 159 Sulfamethazine . 96, 154 Sulfamethoxazole . 156-157 Sulfanilamide . 97, 127, 144, Sulfathiazole . 97, 154-157 Sulfisoxazole . 156-157 2, 4, Tamoxifen citrate . 162-163 Taxol . 116-117 Terconazole . Terfenadine . 164-165 Testosterone acetate . 166 Testosterone benzoate . 166 Testosterone enanthate I.S. ; . 166 Testosterone Propionate I.S. ; . 35, 166 Tetracaine . Tetracycline 167-168 Theobromine . 45, 140, Theophhlline . 45, 140, Thiamine Hydrochloride . 180-181 Toluamide . 160 2, 4, . Trans-Retinol Vitamin A ; 179 Trazodone . 21, Triamcinolone . 122-123 Trimethoprim . 158-159 Trimipramine . 173, 175 Tripelennamine . 176 Triprolidine HCL . 87, 138 Uracil . 149, 179 Valerophenone . 147-148 Verapamil . 29, 177-178 Vitamins . 179-181 Warfarin . Zidovudine . 182 and albenza.

Blood glucose and serum insulin concentrations, mean arterial blood pressure MAP ; and total pancreatic blood flow PBF ; 3, 10 or 20 min after intravenous administration of 1 ml saline or 30% glucose. Some of the animals were also given an intravenous injection of saline or theophylline 6 mg kg body weight ; , whilst others were subjected to a sub-diaphragmatic vagotomy. Blood glucose Serum insulin MAP PBF mmol l ; ng ml ; min x g pancreas ; 3 minutes after glucose or saline Saline + saline 6.6 0.2 1.69 Saline + glucose 22.9 1.6 * 10.18 2.10 * 108 6 0.66 Vagotomy + saline 6.8 0.3 1.75 Vagotomy + glucose 19.0 0.7 * 6.70 1.98 * 118 6 * 0.37 0.05 Tgeophylline + saline 6.9 0.2 1.89 Hteophylline + glucose 23. 0 0.5 * 11.24 1.80 * 110 6 0.86 min after glucose or saline Saline + saline Saline + glucose Vagotomy + saline Vagotomy + glucose Tjeophylline + saline Theophylline + glucose 5.1 0.2 12.7 * 7.8 0.4 19.2 0 0.6 * 2.01 0.30 6.78 * 1.81 0.23 6.07 * 1.59 0.32 5.96 * 103 5 111.

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