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And methylergonovine such as cafergot, migranal, dhe 45, ergotrate maleate, and methergine, as well as halcion, hismanal, orap, propulsid, seldane.
Chronically instrumented pre-term fetal sheep 104 days, term is 147 days ; were studied before and after profound hypoxia induced by complete umbilical cord occlusion for 25 min. An adapted telemetry based implantable nerve was used to record continuous RSNA in 3 fetuses, without paralysis or Notes, for example, side effects of cafergot.
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Community activist J. McDonald Williams and his wife, Ellen, have given $100, 000 to establish the J. McDonald and Ellen Williams Fund for Clinical Care and Service. It will help the medical center develop and implement model systems of clinical care and delivery to medically underserved areas of Dallas. "I've always been interested in helping improve opportunities for Dallas' low.
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Je, Naweza Kutumia Amprenavir Na Dawa Nyingine? Amprenavir inaweza kuleta madhara kadhaa ikitumiwa pamoja na dawa nyingine. Ni muhimu kumweleza daktari wako au mfamasia wako juu ya dawa nyingine zote ulizoandikiwa na unazotumia, ikiwa ni pamoja na vitamini na dawa za mitishamba ; . Amprenavir isitumike pamoja na: Halcion triazolam ; , Versed midazolam ; , Hismanal astemizole ; , Seldane terfenadine ; , Prepulside cisapride ; , Cordarone amiodarone ; , Quinidine, Rifampin na dawa za maumivu makali ya mara kwa mara ya kichwa - migraine mfano Ergomar, Cafwrgot ; . Vitamini E hupatikana kwenye amprenavir. Kuepuka kuzidisha matumizi ya.
While gender made only a slight difference to the proportions of people who felt able to ask questions there were considerable differences between diagnoses Table 2.8 ; . For example, 70% of people with a diagnosis of bipolar disorder felt able to ask questions compared to just over 44% for people with a diagnosis of personality disorder again based on a fairly small sample ; . Table 2.8 Felt able to ask questions? - Diagnosis Diagnosis * Depression Schizophrenia Anxiety Disorder Bipolar Disorder Personality Disorder 2.7 Joint decision making Half of all respondents reported that they had been very or fairly happy that the prescription of their drug had been a joint decision between them and the person making the prescription. As we saw earlier in comments, many people seemed to take the view that either the doctor `knew best', or reported being too unwell to genuinely participate. Table 2.9 How happy were you that the prescription was a joint decision? How happy joint decision Very happy Fairly happy Neither happy nor unhappy Fairly unhappy Very unhappy Number % 157 214 182 Yes 212 106 86 % 59.7 64.6 55.5 No 114 49 52 and capoten, for example, headache.
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Cant time was used to cross-reference and interpret data that were entered as free text and could thus be entered in multiple ways. For example, spelling errors and acronyms as entered by the provider ICD-9, allergies ; were matched to core terms and were eliminated from the search if they could not be interpreted. Laboratory results were `cleaned' by cross-referencing abbreviations and different numerical laboratory units. Discrepancies in key values or errors in data entry resulted in elimination of these items from the search for that record. Approximately 5% of the records were not used due to an inability to `clean' the data fields. The M2D2TM Micromedex; Denver, CO ; medical data dictionary examined the medical terms in the visit notes that contained acronyms, spelling errors, dialects, synonyms and abbreviations in order to provide uniformity and cross-referencing. However, since a natural language processor was not used, similar continuity could not be applied to the standard text in the visit note. Negative values `not', `no', `rule out' ; when associated with potential adverse terms i.e. `no cough', `not swollen', etc. ; resulted in the removal of that sentence from the search process for an incident. This resulted in the elimination of about 10% of the records. A computer program was then developed to query and narrow the data in order to identify incidents or potential ADEs based on four search methods: ICD-9 codes, allergy rules, computer event monitoring rules, and an automated chart review using a term search of the EMR. Incidents were subsequently evaluated to determine whether an ADE was present by evaluating the chart; those results will be presented elsewhere unpublished data ; . The remainder of this paper will describe the four search methods used to identify these potential ADEs.
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However, most trials ended early because the drugs' benefits were so clear.
Dowson AJ, et al. Curr Med Res Opin 2002; 18: 41439. Bronfort G, et al. Cochrane Database Syst Rev 2004; 3 ; : CD001878. Lipton RB, et al. JAMA 2000; 284: 2599605. NSW Therapeutic Assessment Group. Prescribing guidelines for primary care clinicians: migraine last revised 2002 ; , accessed 1 December 2004, clininfo.health.nsw.gov.au nswtag publications guidelines Migraine4 12 02 . Matchar DB, et al. Evidence-based guidelines for migraine headache in the primary care setting: pharmacological management of acute attacks. American Academy of Neurology, 2000, accessed 21 October 2004, aan professionals practice pdfs gl0087 . Lipton RB, et al. Arch Intern Med 2000; 160: 348692. Lipton RB, et al. Cephalalgia 2004; 24: 32132. The Multinational Oral Sumatriptan and Caferg9t Comparative Study Group. Eur Neurol 1991; 31: 31422. Cady RK, et al. Clin Ther 2000; 22: 103548 and carvedilol.
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Do not take ATRIPLA if you are taking the following medicines because serious and lifethreatening side effects may occur when taken together: Hismanal astemizole ; , Propulsid cisapride ; , Versed midazolam ; , Halcion triazolam ; , or ergot derivatives for example, Wigraine and Caferggot ; . In addition, ATRIPLA should not be taken with: Combivir lamivudine zidovudine ; , Emtriva emtricitabine ; , Epivir or Epivir-HBV lamivudine ; , EpzicomTM abacavir sulfate lamivudine ; , Sustiva efavirenz ; , Trizivir abacavir sulfate lamivudine zidovudine ; , Truvada emtricitabine tenofovir disoproxil fumarate [DF] ; , or Viread tenofovir DF ; , because they contain the same or similar active ingredients as ATRIPLA. Vfend voriconazole ; should not be taken with ATRIPLA since it may lose its effect or may increase the chance of having side effects from ATRIPLA. Fortovase, Invirase saquinavir mesylate ; should not be used as the only protease inhibitor in combination with ATRIPLA. Taking ATRIPLA with St. John's wort Hypericum perforatum ; is not recommended as it may cause decreased levels of ATRIPLA, increased viral load, and possible resistance to ATRIPLA or cross-resistance to other anti-HIV drugs.
You should also not take parlodel if you are allergic to it or any other drugs containing ergot alkaloids, such as bellergal-s and cafergot and ciprofloxacin.
Table 1 Cell sizes of V. harveyi wild-type and cgt A mutant strains growing under normal conditions.
It can diagnosed by detecting glucocerebrosidase activity in leukocytes, fibroblasts, or urine in a study from the shaare zedek medical center, researchers evaluated the impact of a program used to screen for carrier status in nearly 30, 000 people and clarinex.
Fraud and abuse by providers, members, contractors, employees, etc. hurts everyone AmeriChoice of New York, the New York State Medicaid program, taxpayers and others. Your assistance in notifying us and cooperating with any potential fraud and abuse occurrence is vital and appreciated in conjunction with our mutual ongoing efforts to coordinate the most effective health outcomes possible for our members.
Mi carga viral todava es indetectable, as que no creo que el tratamiento haya fracasado. Los efectos secundarios todava son mi preocupacin mayor y tengo deseo de cambiar de terapia. Cmo hago esto? and clindamycin and cafergot, for example, atenolol.
Systematic Reviews run on May 2004 ; Databases covered: Medline, Embase, CINAHL, PsycInfo Cochrace Total hits: 1112 Guidelines run on ; The guideline search used the patient stem. Agency for Health Care Research and Quality AHRQ ; ASERNIP-S Canadian Medical Association Centre for Clinical Effectiveness Australia ; Health Technology Assessment NHS Centre for Reviews and Dissemination NHS Quality Improvement Scotland National Institute for Clinical Excellence NICE ; National Electronic Library for Health National Guideline Clearinghouse National Health and Medical Research Council NHMRC ; New Zealand Guidelines Group Swedish Council on Technology Assessment in Health Care SBU ; TRIP Database US National Institutes of Health Total hits: 42 Guidelines evaluated using AGREE instrument: Databases covered: Dates covered: 1999-2004.
Notes age, gender, race and ethnicity: there is insufficient evidence to know if the drugs reviewed in this report differ in their effectiveness or safety with regard to age, gender or ethnicity and clobetasol.
BLOCADREN 20 MG TABLET BLOCADREN 5 MG TABLET BRETHINE 2.5 MG TABLET BRETHINE 5 MG TABLET BREVICON 28 TABLET BROMFED CAPSULE BROMFED-PD CAPSULE BUMEX 0.5 MG TABLET BUMEX 1 MG TABLET BUMEX 2 MG TABLET BUSPAR 10 MG TABLET BUSPAR 15 MG TABLET BUSPAR 30 MG TABLET BUSPAR 5 MG TABLET CAFERGOT TABLET CALAN 120 MG TABLET CALAN 40 MG TABLET CALAN 80 MG TABLET CALAN SR 120 MG CAPLET SA CALAN SR 180 MG CAPLET SA CALAN SR 240 MG CAPLET SA CAPOTEN 100 MG TABLET CAPOTEN 12.5 MG TABLET CAPOTEN 25 MG TABLET CAPOTEN 50 MG TABLET CAPOZIDE 25 15 TABLET CAPOZIDE 25 TABLET CAPOZIDE 50 15 TABLET CAPOZIDE 50 25 TABLET CARAFATE 1 GM TABLET CARDENE 20 MG CAPSULE CARDENE 30 MG CAPSULE CARDIZEM 120 MG TABLET CARDIZEM 30 MG TABLET CARDIZEM 60 MG TABLET CARDIZEM 90 MG TABLET CARDIZEM CD 120 MG CAPSULE CARDIZEM CD 180 MG CAPSULE CARDIZEM CD 240 MG CAPSULE CARDIZEM CD 300 MG CAPSULE CARDIZEM SR 120 MG CAPSULE CARDIZEM SR 60 MG CAPSULE.
HEALTH POLICY W11: ISSUES IN DRUG PRICING, REIMBURSEMENT, AND ACCESS IN THE ASIAPACIFIC REGION Auditorium Chinese translation available ; Discussion Leaders: Stuart O. Schweitzer PhD, Professor of Health Services, UCLA School of Public Health; Yingyao Chen PhD, Professor of Hospital Management, Fudan University School of Public Health.
Race formal pharmacokinetic studies for race have not been conducted.
Hardly comes as Figure 36: Proportion of men reporting having had a surprise, as the sex with men in previous 12 months 100 per cent condom policy is not per cent ; implemented at 22.9 100 per cent, 25 1995 with the result 20 that, while the 15 1996 client who uses 10 9.9 10.6 condoms is pro3.9 5 2.6 tected, the sex 1.0 0 worker is still at military conscripts labourers secondary school risk from those students clients who do not use condoms. difficult to remain abreast of the new Given the high mobility of commer"spots". However, while the "indirect" cial sex workers, they may have commercial sex activity is a source of acquired the infection in another real concern, customer turnover is province ; . likely to be much lower than in commercial sex establishments, leading to In addition to commercial sex estabrelatively less new infections. lishments, sex workers increasingly offer their services in some restaurants, karaoke Figure 37: Proportion of reported consistent condom bars, traditional massage parlours use by men when having sex with men in previous and motels. The 12 months province has no per cent ; known commercial sex establish- 100 1995 ments offering services by males. 1996 41.2 37.5 Men would pick 37.5 1997 23.5 up young men in karaoke bars and 0 restaurants. This military conscripts labourers secondary school students is a changing scene and it is, for instance, lisinopril.
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Skin rashes can occur in patients taking LEXIVA. Rarely, rashes were severe or life threatening. Opportunistic infections can develop when you have HIV and your immune system is weak. It is very important that you see your healthcare provider regularly while you are taking LEXIVA to discuss any side effects or concerns. Most common side effects in clinical studies were diarrhea, headache, nausea, rash, and vomiting. In most cases, these side effects did not cause people to stop taking their medicine. Drug Interactions LEXIVA should not be taken with: AGENERASE amprenavir ; , Halcion triazolam ; , ergot medications Cafergot, Migranal, D.H.E. 45, and others ; , Propulsid cisapride ; , Versed midazolam ; , Orap pimozide ; , Zocor simvastatin ; , Mevacor lovastatin ; , Rifadin rifampin ; , Rescriptor delavirdine mesylate ; , or St. John's wort Hypericum perforatum ; . If you are taking Norvir ritonavir ; , you should not take Tambocor flecainide ; , or Rythmol propafenone hydrochloride ; . Serious and or life-threatening events could occur between LEXIVA and other medications, including Cordarone amiodarone ; , lidocaine intravenous only ; , Elavil amitriptyline HCl ; and Tofranil imipramine pamoate ; , tricyclic antidepressants, and Quinaglute quinidine ; . Women who use birth control pills should choose a different kind of birth control. The use of LEXIVA with Norvir ritonavir ; in combination with birth control pills may hurt your liver. Also, birth.
Table 2 distinguishing upper from lower motor neuron lesions feature upper lesion lower lesion reflexes hyperactive diminished or absent atrophy absent * present fasciculations absent present tone increased decreased or absent * may appear with prolonged limb disuse.
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Roche Pharmaceuticals OTC: RHHBY.PK ; and Trimeris, Inc. Nasdaq: TRMS ; are the leaders in the HIV fusion inhibitor category with their leading product Fuzeon. Fuzeon has faced its difficulties because doctors have been reluctant to prescribe Fuzeon because of their apparent concern about patient compliance. The rate of new prescriptions for Fuzeon dropped approximately 50 percent in 2004, yet Fuzeon was able to achieve worldwide sales of $135 million in 2004 + 27% ; despite the reluctances of doctors. There are currently multiple entry or attachment inhibitors in the drug pipeline that are progressing rapidly. Fusion Inhibitors attempt to inhibit viral attachment and thus prevent HIV from entering the human cell. Scientists hope that by targeting particular sites on both the human cell and the HIV virus that serve as receptors when the virus attaches to the human cell they will be able to prevent infection. One of these receptors is known as the CCR5 receptor. The CCR5 receptor helps direct immune cells to damaged or diseased areas of the body. This is how HIV enters and infects T cells. There is currently only one FDA approved fusion inhibitor: Enfuvirtide; T-20; Fuzeon Roche Pharmaceuticals, OTC: RHHBY.PK ; and Trimeris, Inc., Nasdaq: TRMS.
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10 Ibid. 11 Mokdad, Ali H. et al. 2003 ; Prevalence of obesity, diabetes, and obesity-related health risk factors, 2001. JAMA 289: 76-79.12 ibid 13 Fortess EE, et al. 2001 ; Utilization of essential medications by vulnerable older people after a drug benefit cap: importance of mental disorders, chronic pain, and practice setting. Journal of the American Geriatric Society. Jun; 49 6 ; : 793-7. 14 Soumerai SB, et al. 1991 ; Effects of Medicaid drug-payment limits on admission to hospitals and nursing homes. New England Journal of Medicine. 325 15 ; : 1072-7. 15 The Diabetes Control and Complications Trial Research Group. 1993 ; The effect of intensive treatment of diabetes on the development and progression of long-term complications in insulin-dependent diabetes mellitus. New England Journal of Medicine Sep 30; 329 14 ; : 977-86. 16 Ibid. 17 Tennessee has recently proposed creating a preferred drug list for all seven of its Medicaid managed care plans Kaiser Daily Health Policy Report, 5 6 03.
Even though one in five adults do experience the symptoms typical of IBS, only a handful of people go to see the doctor in order to get professional help. People sometimes tend to think that these symptoms are just indicative of an ordinary bowel problem, but it's not. It is definitely important to seek medical advice if you think you have IBS, for example, side effects of cafergot.
| Cafergot compositionAnd neuropathy. Generalized vascular damage in insulin-dependent diabetic patients. Horm. Metab. Res. Suppl. 26, 68-70. 54. Hayes B.E., Clarke, D.E. 1990 ; Semicarbazide-sensitive amine oxidase activity in streptozotocin diabetic rats. Res. Commun. Chem. Pathol. Pharmacol., 69, 71-83. 55. Kurkijarvi R., Adams D.H., Leino R., Mottonen T., Jalkanen S., Salmi M. 1998 ; Circulating form of human vascular adhesion protein1 VAP-1 ; : increased serum levels in inflammatory liver diseases. J. Immunol. 161, 1549-1557. 56. Mercier N., Moldes M., El-Hadri K., Feve B. 2001 ; Semicarbazidesensitive amine oxidase activation promotes adipose conversion of 3T3-L1 cells. Biochem. J., 358, 335-342. 57. Morin N., Lizcano J.M., Fontana E., Marti L., Smih V., Rouet P. 2001 ; Semicarbazide- sensitive amine oxidase substrates stimulate glucose transport and inhibit lipolysis in human adipocytes. J. Pharmacol. Exp. Ther. 297, 563-572. 58. El Hadri K., Moldes M., Mercier N., Andreani M., Pairault J., Feve B. 2002 ; Semicarbazide-sensitive amine oxidase in vascular smooth muscle cells. Differentiation-dependent expression and role in glucose uptake. Arterioscler. Thromb. Vasc. Biol., 22, 89-94. 59. Langford S.D., Trent M.B., Boor P.J. 2002 ; Semicarbazide-sensitive amine oxidase and extracellular matrix deposition by smoothmuscle cells. Cardiovasc. Toxicol., 2, 141-150. 60. Yoong K.F., McNab G., Hbscher S.G., Adams D.H. 1998 ; Vascular adhesion protein-! and I-CAM-1 support the adhesion of tumor-intfiltrating lymphocytes to tumor endothelium in human hepatocellular carcinoma. J. Immunol., 160, 3978-3988. 61. Souto R.P., Vallega G., Wharton J., Vinten J., Tranum-Jensen J., Pilch P.F. 2003 ; Immunopurification and characterization of rat adipocyte caveolae suggest their dissociation from insulin signalling. J. Biol. Chem., 278, 18321-18329. 62. Palgreyman M.G., McDonald I.A., bey P., Danzin C., Zreika M., Cremer G. 1994 ; Haloallylamine inhibitors of MAO and SSAO and their therapeutic potential. J. Neural. Transm. 41 Suppl ; , 407-414. 63. Lyles G.A., Marshall C.M.S., McDonald I.A., Bey P., Palfreyfan M.G. 1987 ; Inhibition of rat aorta semicarbazide-sensitive amine oxidase by 2-phenyl-3-haloallylamines and related compounds. Biochem. Pharmacol., 36, 2847-2853. 64. Morikawa F., Ueda T., Arai Y., Kinemuchi H. 1986 ; Inhibition of monoamine oxidase A-form and semicarbazide-sensitive amine oxidase by selective and reversible monamine oxidase inhibitors amiflamine and FLA 788 + ; . Pharmacology, 32, 38-45. 65. Arai Y., Toyoshima Y., Kinemuchi H. 1986 ; Studies on monoamine oxidase and semicarbazide sensitive amine oxidase. Part II. Inhibition by a-methylated substrate-analogue monoamines, a-methyltriptamine, a-methylbenzylamine and two enanthiomers of a-methylbenzylamine. Jpn. J. Pharmacol., 41, 191-197. 66. Kinemuchi H., Jinbo M., Tabata A., Toyoshima Y., Arai Y., Tadano, T. 2000 ; 2-Bromoethylamine, a suicide inhibitor of tissuebound semicarbazide-sensitive amine oxidase. Jpn. J. Pharmacol., 83, 164-166. 67. Lyles G.A. 1995 ; Substrate specificity of mammalian tissuebound semicarbazide-sensitive amine oxidase, Yu P.H., Tipton K.F., Boulton A.A. Eds ; , Current Neurochemical and Pharmacological Aspects of Biogenic Amines. p. 293-303, Elsevier, Utrecht. 68. Lizcano J.M., deArriba A.F., Tipton K.F. 1996 ; Inhibition of lung semicarbazide-sensitive amine oxidase SSAO ; by some hydrazine derivatives. Biochem. Pharmacol., 52, 187-195. 69. Yu P.H., Zuo D-M. 1997 ; Aminoguanizine inhibits semicarbazide-sensitive amine oxidase activity: implication for advanced glycation end product and diabetic complications. Diabetologia, 363, 1243-1250. 70. Holt A., Callingham B.A. 1995 ; Further studies on the ex vivo effects of procarbazine and monomethylhydrazine on rat semicarbazide-sensitive amine oxidase and monoamine oxidase activities. J. Pharm. pharmacol., 47, 837-845.
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PEL cells. As a consequence, the tumors appear very homogenous, highly angiogenic, and maintain PEL morphology Fig. 1 ; . Whether the growth factor-depleted extracellular matrix additionally provides signals through direct contact with the cells as occurs during normal development is currently unresolved. KSHV was retained in the PEL tumor cells as evidenced by uniform LANA expression at both the mRNA and protein levels Figs. 3 and 4 ; . Without Matrigel, PEL did not grow in the s.c. environment within the time frame of our observation period. Rather, rare variants emerged after a very long in vivo incubation. They were characterized by disorganized growth, local necrosis, and altered viral gene transcription Figs. 2 4 ; and, hence, would not be expected to mimic the human disease. We found substantial evidence for KSHV lytic gene expression in PEL grown in the mouse xenograft model Fig. 4 ; . These data substantiate a paracrine modus operandi for KSHV pathogenesis that postulates that KSHV lytic genes, such as vGPCR orf74 31, 81, 82 ; , in a portion of tumor cells contribute to tumor development as a whole. Presumably, LANA and the other KSHV latent oncogenes suffice to initiate B-cell transformation under optimal growth conditions in the presence of the progrowth environment high endogenous interleukin 6 and chronic antigen stimulation ; of the splenic mantle zone where KSHV-associated MCD can be found. The rich progrowth environment of the mantle zone is mimicked in current suspension cultures of PEL cell lines that include serum growth factors and endogenous, human interleukin 6. In contrast, KSHV lytic oncogenes would be needed to sustain ectopic pleural growth in advanced PEL. This dichotomy is mimicked in our mouse xenograft model by i.p. or s.c. plus Matrigel implantation. Studies to identify changes in cellular gene expression between these two microenvironments are currently ongoing. We found increased transcription of KSHV lytic genes in the BCBL-1 xenografts Fig. 4 ; . This prompted us to investigate the susceptibility of such tumors to the antiviral drug ganciclovir, which requires activation by the viral thymidine kinase TK orf21 ; and or the viral phosphotransferase PT orf36; Refs. 79, 83 ; . Ganciclovir inhibits KSHV replication in BCBL-1 cells and SCID-hu mice 55, 64, 71, ; . Investigations of the effect of ganciclovir on KSHVassociated PEL are warranted because the treatment of EBV-associated lymphomas with antiviral regimens has received renewed attention recently due to the predicted high therapeutic index and antitumor activity of these virus-specific drugs 69, 79, 84 ; . Both EBV and KSHV encode a viral thymidine kinase BXLF1 and orf21, respectively ; , which can phosphorylate ganciclovir, although the efficiency of this reaction compared with the thymidine kinase of the and herpesviruses is still under debate 79, 83, 86, ; . In the case of KSHV, the selectivity index for ganciclovir is 39 77 ; Therefore, we hypothesized that the presence of these viral enzymes should results in the selective growth retardation of KSHV-infected cells reviewed in Ref. 92 ; . Furthermore, we hoped that, as in the case of combination nucleoside gene-therapy in glioblastoma 93 ; where not every tumor cell needed to express the viral kinase, a significant bystander effect would be observed for PEL tumors. Contrary to our expectations, ganciclovir alone did not significantly inhibit tumor growth in the BCBL-1 xenograft model Table 1 ; nor did it kill BCBL-1 cells after KSHV reactivation in culture Fig. 6 ; . Whole viral genome profiling showed that at the IC90 ganciclovir allowed for the sustained high level transcription of viral early genes by blocking completion of the viral life cycle. This suggests that the clinical effects of ganciclovir on KS as seen in a study of human CMV-induced retinitis 78 ; may be due to its systemic inhibition of KSHV lytic replication or reactivation, which can be caused by human cytomegalovirus 62 ; , but not necessarily a direct antitumor effect. Alternatively, KS lesions may exhibit a still greater proportion of cells expressing lytic genes or are.
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