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ICD-9-CM Table of Drugs and Chemicals FY07 ; PoisonAcciSubstance ing dent Strobane Strophanthin Strophanthus hispidus or kombe Strychnine rodenticide ; salts ; medicinal NEC Strychnos ignatii ; -see Strychnine Styramate Styrene Succinimide anticonvulsant ; mercuric-see Mercury Succinylcholine Succinylsulfathiazole Sucrose Sulfacetamide ophthalmic preparation Sulfachlorpyridazine Sulfacytine Sulfadiazine silver topical ; Sulfadimethoxine Sulfadimidine Sulfaethidole Sulfafurazole Sulfaguanidine Sulfamerazine Sulfameter Sulfamethizole Sulfamethoxazole Sulfamethoxydiazine Sulfamethoxypyridazine Sulfamethylthiazole Sulfamylon Sulfan blue diagnostic dye ; Sulfanilamide Sulfanilylguanidine Sulfaphenazole Sulfaphenylthiazole Sulfaproxyline Sulfapyridine Sulfapyrimidine Sulfarsphenamine Sulfasalazine Sulfasomizole Sulfasuxidine Sulfinpyrazone Sulfisoxazole ophthalmic preparation Sulfomyxin Sulfonal 989.2 972.1 988.2 E863.0 E858.3 E865.4 E863.7 E854.3. Both assessing the dangerousness of an activity and determining how much danger is acceptable will become the exclusive domain of each of my kids as it pertains to them, for example, nimodipine nimotop.
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UNIT FIVE HEALTH PROMOTION AND RISK ASSESSMENT . 235.

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There have been reports of patients developing painful kidney stones while on HIV drug regimens containing Reyataz. While little is known about the frequency of the kidney stones, or if certain risk factors are involved, consuming eight glasses of water a day while taking Reyataz may help decrease the risk of this possible side effect. Changes in the way your heart beats may occur when taking Reyataz. If you get dizzy or lightheaded these could be symptoms of a heart problem. Other possible side effects of Reyataz include headaches, pain tingling in the arms and legs, nausea, diarrhea, abdominal discomfort, and rash. Reyataz may not increase fat levels in the blood--a symptom of lipodystrophy--like other protease inhibitors. In a few studies comparing Reyataz to other protease inhibitors, those receiving Reyataz had no increases in triglycerides which can cause pancreatitis ; or "bad" LDL cholesterol, which can increase the risk of heart disease or stroke. There were, however, significant increases in HDL "good" ; cholesterol, which can help reduce the risk of heart disease. It is still not known if Reyataz can cause lipodystrophy's body-shape changes, such as an increase in fat around the gut or a loss of fat in the arms, legs, or face. It is also not known if HIV-positive people who continue taking Reyataz for a long period of time e.g., longer than one year ; will be able to keep their cholesterol and triglyceride levels low. Side effects other than those listed here may also occur. Talk to your doctor about any side effect that seems unusual or that is especially bothersome, because vademecum. Within the formulary requirements; such usages trigger prior authorization procedures requiring the prescribing physician to justify the use of the medication.20.

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Your health insurance may or may not cover some of these modalities and nimodipine.
Montclair Baptist Medical Center 880 Montclair Road, First Floor Birmingham, AL 35213 205 599-3500 Cooper G. Hazelrig, MD, FACC William R. Stetler, MD, FACC James M. Jones, III, MD, FACC Richard O. Russell, Jr., MD, FACC Jerry W. Chandler, MD, FAC William R. Harrison, MD, FACC Eric D. Cohen, MD, FACC Russell C. Reeves, MD, FACC W. Hansford Watford, Jr., MD, FACC James R. Boogaerts, MD, FACC Robert I. Brock, MD, FACC John T. Eagan, Jr., MD, FACC Donald G. Gordon, MD, FACC Paul J. Troup, MD, FACC Percy J. Colon, III, MD, FACC Luiz Pinheiro, MD Raashid Ashraf, MD, FACC Chris Y. Kim, MD Brookwood Medical Center 2022 Medical Center Drive, Suite 510 Birmingham, AL 35209 205 877-9290 Larry E. Dye, MD, FACC Anderson M. Morris, MD, FACC Michael B. Honan, MD, FACC David H. Jackson, MD, FACC Steven E. Jones, MD, FACC Dianne Barnard, MD, FACC Elizabeth E. Branscomb, MD, FACC Paschal E. Redding, III, MD, FACC Anuradha V. Rao, MD Peter P. Scalise III, MD, FACC Shelby Baptist Medical Center 1022 North 1st Street, Suite 500 Alabaster, AL 35007 205 663-5775 C. Dale Elliott, MD, FACC John D. McBrayer, MD, FACC Michael E. McKinney, MD, FACC Mark L. Mullens, MD, FACC Joyce R. Koppang, MD, FACC Princeton Baptist Medical Center 817 Princeton Avenue SW, Suite 202 Birmingham, AL 35211 205 786-8815 A.M. Reddy, MD, FACC Vasu Goli, MD, FACC Coosa Valley Baptist Medical Center 209 West Spring Street, Suite 104 Sylacauga, AL 35150 256 245-5833 Robert F. Ingram, MD, FACC Walker Baptist Medical Center 3400 Highway 78 East, Suite 311 Jasper, AL 35501 205 221-9494 Richard I. Kim, MD, FACC HealthSouth Medical Center 1201 11th Avenue South, Suite 301 Birmingham, AL 35205 Anderson M. Morris, MD, FACC Outreach Locations Hospital Coverage Childersburg - Clanton Columbiana - Dora Hamilton - Oneonta Pell City - Talladega Physician Referral Line 800 676-9358.
NEO-SYNEPHRINE . Phenylephrine NEPTAZANE . Methazolamide NESTABS . Vitamins, prenatal NETROMYCIN . Netilmicin NEULASTA . Pegfilgrastim NEUPOGEN . Filgrastim NEURONTIN . Gabapentin NEUTREXIN . Trimetrexate NEVANACTM . Nepafenac NEXAVAR . Sorafenib NEXIUM . Esomeprazole NIACOR . Niacin NIASPAN . Niacin, extended-release NICLOCIDE . Niclosamide NICODERM CQ Nicotine, transdermal NICORETTE . Nicotine, chewing gum NICOTROL INHALER . Nicotine, inhaler NICOTROL NS Nicotine, nasal spray NICOTROL TRANSDERMAL . Nicotine, transdermal NIFEREX . Iron as polysaccharide-iron complex ; NIFEREX-150 FORTE . Iron as polysaccharideiron complex ; + Folic acid + Vitamin B12 NIFEREX-PN Vitamins, prenatal NILANDRON . Nilutamide NIMOTOP . Nimodipine NIPRIDE . Sodium nitroprusside NIRAVAMTM . Alprazolam, orally-disintegrating tablet NISOCOR . Nisoldipine, extended-release NITRO-BID Nitroglycerin, sustained release NITRO-DUR Nitroglycerin, transdermal NITROLINGUAL . Nitroglycerin, sublingual spray NITROSTAT . Nitroglycerin, sublingual tablets NIX . Permethrin NIZORAL . Ketoconazole shampoo NOLVADEX . Tamoxifen NORCO . Hydrocodone + Acetaminophen NORCURON . Vercuronium NORDETTE . Ethinyl estradiol + Levonorgestrel NORDITROPIN . Somatropin NORFLEX Orphenadrine Citrate, extended-release NORGESIC . Orphenadrine Citrate + aspirin + caffeine NORINYL 1 + 35 Norethindrone + Ethinyl estradiol NORINYL 1 + 50 Norethindrone + Mestranol NORITATE . Metronidazole NORMODYNE . Labetalol NOROXIN . Norfloxacin and noroxin.
INRUD is a co-operative organisation of health workers, administrators and researchers in developing countries aiming to improve drug use. There are several different national groups. WHO Drug Information. Subscription. Published in English and French by the World Health Organization, 12 11 Geneva 27, Switzerland. Journal of drug development and regulation. WHO CDD Programme. Further reading Chetley, A, - 1987. Antibiotics: the wrong drugs for diarrhoea. Health Action International. Available from HA1 Europe. Cutting, WA M, 1989. Selfpresctibing andpromotion of antidiarrhoeal drugs. Lancet 8646: 1080. Dean, P and Ebrahim, G J, 1986. Practical care of sick children: a manual for use in small tropical hospitals. Macmillan, 348 pages. Ghai, 0 P, 1987. Understanding and.

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Medications. These medications themselves can cause stomach bleeding and ulcers, so using hydrochloric acid with them increases the risk. Keep in mind that this particular benign, natural solution involves replacing something the body is missing. It avoids using synthetic chemicals the body is ill-equipped to handle and norfloxacin. Over the past several years, diagnostic imaging devices have become increasingly popular tools in clinical practice for diagnosing and managing glaucoma. Scanning laser polarimetry, scanning laser topography and optical coherence tomography have all gone through several iterations to give us the GDx, Heidelberg Retinal Tomograph III and the Stratus OCT III, respectively. The number of doctors using one or more of these devices increases each year. While the instruments have been proven accurate in assessing tissue structure, laser imaging devices represent only one piece of the diagnostic puzzle. This article is not intended to discuss the relative merits of the various imaging devices for the diagnosis of glaucoma. Clearly, they each have unique abilities to assess structure. Each has advantages and limitations, and no one device enjoys the reputation of being appropriate for every patient in every situation. Likely, the information provided by each is complementary to that of the others. Regardless of the device or technology used, however, the challenge is to correctly interpret the data and properly apply it in the context of the overall clinical situation. Failure to do so can result in misdiagnosis and poor patient care. Unfortunately, too many doctors consider the diagnostic imaging devices to be "Silicon Valley Rumplestilskins" that spin gold from straw. We must consider exactly what the data tells us. As an example, consider the GDx printout below. While scanning laser polarimetry is used as an example here, the principles remain the same across all technologies. Each device attempts to use parameters that are most sensitive in separating normal patients from those with glaucoma. The most pertinent question is: Does this patient have glaucoma? Clearly, there is an asymmetric nerve fiber layer NFL ; between the two eyes that appears worse in the left eye. There are numerous departures from the normative database in each eye of high statistical significance. Furthermore, the majority of the parameters fall well outside normal limits, especially in the left eye. Finally, the Nerve Fiber Indicator NFI, an experimental indicator of the likelihood of glaucoma being present ; is 36 in the right eye and 62 in the left eye. These numbers are typically found in patients with glaucoma. This brings us back to the original question: Does this patient have glaucoma? Unfortunately, we cannot and should not ; try to make a clinical decision based on only one piece of information. It is important to understand what this printout actually tells us. It is not telling us that the patient has glaucoma. In fact, it is not directly telling us that an abnormal NFL is present. What it is actually telling us is that the measurement of the NFL in each eye deviates from the expectation for normal as described by the normative database at levels that are statistically significant--nothing more. Now, interpretation is required. In most normal patients there will be, just by chance, abnormalities that are statistically significant. What becomes important is the ability to discern statistically significant abnormalities from those that are clinically significant. This ability comes from experience derived from performing these technologies on many thousands of patients with various levels of disease. In the example presented here, the statistically significant departures from the normative database are also, in our collective opinion, clinically significant, because we rarely see abnormalities such as these in normal patients. We are not saying these deviations are abnormal; rather that we rarely see them in normal patients. This impression comes from clinical experience with thousands of patients. Thus, our interpretation of this study, strengthened by personal clinical experience, is that this analysis is consistent with an abnormal NFL in each eye. Does this mean that the patient has glaucoma? The measured NFL thickness deviates from the normal population represented in the device's normative database at a high level of statistical significance. Based upon our experience, this pattern of departure from the normative database is rarely seen in normal patients. However, this does not conclusively mean that the patient has glaucoma. Other conditions, such as optic disc drusen, disc hypoplasia and other congenital anomalies, can affect the NFL. Thus, we would be remiss if we simply diagnosed glaucoma based upon an interpreted abnormal NFL without considering any other factors, such as IOP, disc appearance, family history, medical history, visual field analysis or corneal thickness. By measuring and comparing the patient's NFL to a normative database, we learn from our. Incidence and prevalence AUB can account for 10% of visits to family physicians and PAs2, 3 and approximately 5% of visits to general practitioners annually.1 Most cases occur 5 to 10 years before menopause or after menarche. A common reason for referral from primary to secondary care, AUB comprises one third of all outpatient gynecology visits.4 Up to 20% of women with DUB will seek consultation.2 DUB affects approximately 5% of menstruating women, comprising about 80% of cases of menorrhagia, the most common cause of iron-deficiency anemia in the developed world.5 Unfortunately, most primary care practitioners do not understand the pathophysiology and principles of management of DUB, although the disorder can be managed in primary care. Etiology Interaction among the hypothalamus, anterior pituitary, ovaries, and endometrium govern the menstrual cycle see Figure 1 ; . Dysfunction in this system can reThe author works in adult medicine at Community Health Care, Inc, Davenport, Iowa. She has indicated no relationships to disclose relating to the content of this article and nateglinide.

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Sanford I. Weill Medical College and Graduate School of Medical Sciences.

Jan 31, 2006 30, alert ; nimodipino ; nimodipinum ; nimotop; periplum drug category : nimotop is categorized under the following by the fda: vasodilator agents; antihypertensive agents; vasodilator agents; atc: c08ca06 dosage forms : capsule absorption : not available interactions : drugbank: interactions for nimodipine interactions for nimodipine: it is possible that the cardiovascular action of other calcium channel blockers could be enhanced by the addition of nimotop ò and viramune.

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And it evolves to grow resistance to flu drugs, for instance, prednisone. Exposure to silica dust, radiographic evidence of silicosis, and `the absence of any good reason to believe that the radiographic findings are the result of some other condition.' It is also important that the time between exposure and the onset of disease is consistent with the latency period typical of silicosis. Pls.' Informational Br. Regarding Diagnosis Silicosis at 2 citing Hans Weill, et al., Silicosis and Related Diseases, in OCCUPATIONAL LUNG DISORDERS 286 3rd ed. 1994 Daniel E. Banks, Silicosis, in TEXTBOOK OF OCCUPATIONAL AND ENVIRONMENTAL MEDICINE 380-81 2nd ed. 2005 ; . ; The testimony of the diagnosing doctors was in accord with the above summary. For instance, one of the Plaintiffs' diagnosing doctors, Dr. Jay Segarra, a pulmonologist and NIOSH-certified Breader practicing in Biloxi, Mississippi, elaborated as follows about the generally-accepted methodology for diagnosing silicosis: [T]he diagnosis of [silicosis] rests on, basically, three factors. One is an appropriate chest X-ray and I'll tell you what that means in a minute. An adequate exposure history which I'll explain in a minute. And finally, the absence of any other disease that would be more likely to explain the radiographic findings or clinical symptoms or whatever than Silicosis. An appropriate chest X-ray for a B-reader means, at least, primarily small, rounded opacities. They don't all have to be rounded but they should, at least, be primarily rounded. And involving, at least, one of the upper lung zones of an alveoli profusion of 1 0 greater. This is in the absence of some superior medical data that you generally don't have such as a high resolution chest CT scan or a tissue sample where you can look under the microscope. Most of the time, you don't have that available. So, that's the chest X-ray. What an adequate exposure history means is that the physician or an agent of the physician has just got taken from the patient a history of exposure to potentially toxic, environmental substances including organic dust and inorganic dust. And determine that the level of exposure -- the intensity and duration was sufficient to and nortriptyline!

PN were also found in farmer's cheese, but PB was not present. Their concentrations, however, were significantly lower approx. 3.0-4.0 mg 100g ; and did not vary so drastically between batches, as was the case of PG. Some analyses indicated trace amounts of PB, but the concentrations less than a milligram per 100 g ; were at the border of method sensitivity. The results are summarized in Table 1. Our experiments confirmed the results of earlier studies on PG in cow's milk 26 ; . In our studies we used fresh whole milk as well as condensed milk. We found that, similar to the farmer's cheese samples, milk contained mostly PG. The concentration of PG was approximately 3 times higher in the condensed milk than in the fresh milk. The differences in the PG concentrations can be explained simply by the more concentrated sample. The other compounds, i.e. isoPG and A10 were found in trace concentrations less than a milligram per 100 g ; . Nevertheless, PN and PB were not found at all see Table 1 ; . The significantly lower concentrations of isoPG, and A10 can be caused by the process of the milk preparation, such as pasteurization. Interesting results were obtained in the experiment with whey. The amount of PG determined in the whey was comparable to that present in farmer's cheese. These results were obtained for the whey sample, which was concentrated on the rotary evaporator and then deproteinized by addition of acetonitrile. The other compounds, isoPG, A10 and PN were found from trace amounts to 6.0mg 100g, comparable to those present in the other dairy products. The analysis of feta cheese was more difficult to perform due to the high content of fat. PG was in concentrations approximately 7 times lower than in farmer's cheese. This may be explained by the different source of the product sheep vs. cow ; and the loss of some compounds during the preparation steps, especially during the fat extraction!
Media response was fast and furious, " said a funeral home employee whose operation was caught in the media spotlight. "They loved to play on the terms that indicate danger." When Toronto Public Health issued the public notice about Mr. S's visitation it named the funeral home, which was understandable. However, this funeral industry worker said that the media continued to link the home's name with the SARS story long after the quarantine period from the visitation ended. People, including suppliers, avoided the home because the name was still in the media. "The media lacked understanding, " he said. There was a feeling, certainly within BLD, that the BLD connection with SARS was hyped in the media. BLD member Don O'Shaughnessy of Scarborough, who was quarantined during the Easter outbreak, certainly thought so: When you see yourself [BLD] identified in a Time magazine graphic as a locus of the disease, it hurts, especially when the information is wrong.371 He said BLD should have been given the same consideration about privacy as individuals. The community really was singled out and the name BLD was carelessly used372. In fairness, the media had an important duty to report on this serious public health threat. The spread of a deadly disease into the community through any identifiable group, whether it be a religious or ethnic group or a visible minority, is a story that must be covered. The difficulty with reporting such stories is that they are easily sensationalized and require scrupulous accuracy, balance and fairness. The media faced real difficulties in reporting the BLD story. SARS was a new threat and dealing with it was a learning experience. Efforts to get a quick and firm grip on the disease were hampered by a lack of clear facts in the fog of worry over a deadly developing situation. Even the public health authorities, on whom the public and the media were relying for solid information, did not have all the facts. Although the media generally did a good job in SARS, sometimes an outstanding job, there were and pamelor. Sacred Heart Protocol for Hypokalemia * Use dose if SCr 2.5 mg dL IV PO preferred ; 3 1 ; 40 mEq + mEq L 2 ; Check K after 1 hour * Use IV * 3 ; Repeat table 3 3.4 1 ; 20 40 mEq 1 ; 20 mEq q 2hr x 2 mEq L doses 40 mEq total ; 2 ; Check K + after 1 hour 2 ; Check K + after 2 hours 3 ; Repeat table 3 ; Repeat table 3.5 1 ; 20 mEq 1 ; 20 mEq 3.9 2 ; Check K + in morning 2 ; Check K + in morning mEq L.

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A biomarker of exposure is a measure of the level of exposure of an individual to a specific component and, in addition, also encompasses those factors which influence the exposure. There are various ways of measuring exposure with varying degrees of invasiveness. The decision as to which approach is best must take into account factors such as the nature and metabolism of the chemical species, and the level and frequency of exposure. 2.1 Dietary exposure from food One approach to measuring exposure is to measure the levels of specific components in foods. This forms the basis of much of the traditional monitoring process carried out by legislative authorities. For example, in the UK, surveillance programmes exist which focus on a diversity of analytes such as heavy metals, dioxins, polycyclic aromatic hydrocarbons, veterinary drugs, pesticides, mycotoxins and packaging materials. One problem with this type of approach is attempting to define what is meant by an acceptable or "safe" level of contamination. A simple illustration is the measurement of levels of the mycotoxin, aflatoxin B 1. Aflatoxin B1 is produced by the fungus Aspergillus flavus. It is endemic in crops, such as peanuts, pistachio nuts and figs, which are grown in parts of the world where the climate favours the producing organism generally hot and damp conditions ; . Aflatoxin B1 is considered to be a genotoxic carcinogen with the primary site of action being the liver. Definitive proof of its role as a causative agent in heptocellular carcinoma in humans has not yet been established but its classification as a suspected genotoxic carcinogen means that the acceptable level for its presence in food should be the limit of analytical detection currently of the order of 0.1 ppb ; . In fact, such a limit is practically unenforceable since contamination is both widespread at a low and orap and nimotop, because ranitidina. 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J. Discharge Patient to Prior Home Community and Arrange for Medical Follow-Up in Primary Care and pimozide. 24. When a number of people have free access to a resource a pasture to graze their cattle, a fishery, an urban highway ; , each individual has an incentive to use up as much of the resource as possible before other users do the same. Therefore, the commons becomes crowded with cattle or fishermen or cars ; , and the resource grassland, fish stock, highway ; deteriorates in quality as the result of overuse overgrazing, overfishing, traffic congestion ; . Each user has an incentive to use up the resource because he is not fully liable for the cost of doing so. Part of the cost is born by others. None of the users has an incentive to reduce his use the size of his herd, his fishing catch, his highway trips ; or to consider other means of maintaining the resource for example, by supplementing the grass with feed grown on one's private property, by privately farming fish, or by car pooling ; . So all of those with access try to use up the resource before someone else does, and the commons deteriorates and is perhaps even destroyed. Contrast this process with the outcome under private property a private pasture, a fish farm, a private road ; . If an individual owner overuses his resource, he bears the full costs of that action. His resource deteriorates in quality and loses its long-run productive value. Therefore, the private owner has an incentive to conserve his property so that it can be used to generate income or other benefits over a long period. Crowding is not the only consequence of free access, however. When a resource is overused, it deteriorates rapidly in quality and is used up inefficiently, so the quality of the output fatter, healthier cattle, the size of fish, travel time and convenience ; diminishes rapidly over time. This outcome could be offset with appropriate investments in maintenance or improvement the grass might be fertilized or replanted, a fishery might be restocked, or people might car pool ; , but the individuals with common access to the resource have no incentive to invest in maintenance because they cannot exclude others from benefiting from such an investment other people's cattle will consume part of the new grass, other fishermen will catch part of the new fish, and other drivers will add trips on the highway ; . Two characteristics of common-pool resources prevent a Pareto solution. First, because users do not pay for the use of the resource, they tend to overuse it. The costs of this overuse are external to the individual decision makers because such costs are shared with imposed on ; others. Second, because others cannot be excluded from benefits of investing in maintenance or improvements that would increase the productivity of the resource, these benefits are external to the decision maker, and there is an underinvestment in such activities. In essence, the investment in the maintenance of public, common-access property generates external benefits. This process has been called the "tragedy of the commons, " a concept originally attributed to biologist Garrett Hardin 1968 ; . The classic treatment of the subject in economics is by Gordon 1954 ; , but substantial research supports the hypothesis see Libecap 1984; Johnson and Libecap 1982 ; . Also see Benson 1996 for a discussion of the consequences of changes in law that reduce the security of property rights and produce results analogous to those in a commons. 25. When property rights are relatively insecure, bargaining is also less likely Coase 1960 ; . When the insecurity arises because of government's power to take, however, there is an additional reason for expecting bargaining to decline. People who can operate effectively in the political arena essentially have potential claims on other people's property. Seeking control of the desired land through political channels is costly, of course, but if it is expected to be less costly than direct bargaining and voluntary exchange, the incentive to seek involuntary transfers is strong. Thus, individuals who are active in and familiar with the political process are likely to choose that arena because the marginal cost of seeking condemnation is very low once someone has invested in building political connections and influence, whereas individuals who. Middot; nomotop is in the fda pregnancy category this means that it is not known whether nimktop will be harmful to an unborn baby.
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Emergency: Reduce wait times for patients in Emergency Departments while striving to ensure the best possible quality patient care Determine why patients go to Emergency Departments when they don't require that high a level of acute services, and ensure changes can be made to reduce pressure on the department Surgery: Improve patient access to surgeries, including an emphasis on helping to develop a provincial surgical patient registry Implement a common process across IH to ensure timely access to surgery and ensure resources are used efficiently. Emphasis will be on joint replacements. Patient Safety Commit to a culture of quality patient care with transparent processes, educational resources and accountability at all levels Implement recommendations put forth by the Provincial Patient Safety Taskforce Commit to the six patient safety initiatives endorsed by the Board and the Health Authority Medical Advisory Committee, as follows: 1. At the first sign of patient decline: Deploy rapid response teams 2. To prevent avoidable deaths from heart attack: Consistently deliver reliable, evidence-based care for Acute Myocardial Infarction 3. Prevent Adverse Drug Events ADES ; : Implement medication reconciliation 4. Prevent Central Line Infections: Implement a series of interdependent, scientifically grounded steps called the "Central Line Bundle" 5. Prevent Surgical Site Infections: Reliably deliver the correct perioperative antibiotics at the proper time 6. Prevent Ventilator-Associated Pneumonia: Implement a series of interdependent, scientifically grounded steps including the "Ventilator Bundle" Consistent Authority-Wide Standards and Policies Align clinical and administrative practices in accordance with provincial, national and international evidenced-based protocols. HR Recruitment and Retention Redouble efforts to attract excellent clinical and administrative healthcare professionals to prop.

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