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931 Motor and behavioral outcomes after bilateral GPi deep brain stimulation for severe Tourette syndrome . J.Shahed, .J.Poysky, .C.Kenney, .R.Simpson, .J. Jankovic. Houston, .Texas, A ; 932 Tic disorders associated to epilepsy: 2 cases . H.Alonso-Navarro, .T.Adeva-Bartolom, .F.J. Jimnez-Jimnez. Salamanca, .Spain ; 933 Early-onset Tourette syndrome . F.Richer, .P.Lesperance, .S.Chouinard, .G.Rouleau. Montreal, .Quebec, nada ; Tremor Poster.numbers.934-973 934 A new familial disorder: Saccadic oscillations of the eyes . A.G.Shaikh, .K ura, .L.M.Optican, .S.Ramat, .R.J. Leigh, .D.S.Zee. Baltimore, .Maryland, A ; 935 Novel molecular mechanism of essential tremor a computational approach . A.G.Shaikh, .S.Ramat, .L.M.Optican, .K ura, .D.S. Zee. Baltimore, .Maryland, A ; 936 Connexin gap junctions neurophysiological correlate and therapeutic target for oculopalatal tremor . A.G.Shaikh, .S.Hong, .D.Solomon, .K.Liao, .L.M. Optican, .R.J.Leigh, .D.S.Zee. Baltimore, .Maryland, . USA ; 937 The effect of muscle loading on tremor dynamical characteristics in the essential tremor patients . S.Blesic, .J.Maric, .N.Dragasevic, .S lanovic, . V.S.Kostic, .M.R.Ljubisavljevic. Al.Ain, ted.Arab. Emirates ; 938 Effectiveness of piracetam in action tremor myoclonus of patients with Parkinson's disease . R.Neshige. Kurume.City, .Fukuoka, .Japan ; 939 An open label study of pramipexole for the treatment of essential tremor . L.Lay-Son, .D.Saez, .O.Trujillo. Santiago, .Chile ; 940 Disappearance of essential tremor after capsular infarction . N.S.Oztekin, .M.F.Oztekin. Ankara, .Turkey ; 941 Impaired motor speech and balance control in essential tremor . M.Kronenbuerger, .P.Buderath, .B ank, .C omm, . V.A.Coenen, .V.M.Tronnier, .K.L.Kiening, .W.Ziegler, .D. Timmann. Aachen, .Germany ; 942 Essential tremor easy to see, difficult to describe and control . N.Yardimci, .S.Benli. Ankara, .Turkey. Affiliations of authors: Division of Oncology Drug Products RD, JJ, GW, RP ; , Division of Therapeutic Biological Oncology Products PK ; , Center for Drug Evaluation and Research, U.S. Food and Drug Administration, Rockville, MD. Correspondence to: Ramzi Dagher, MD, HFD-150, Center for Drug Evaluation and Research, U.S. Food and Drug Administration, 1451 Rockville Pike, Rockville, MD 20852 e-mail: dagherr cder.fda.gov ; . See "Notes" following "References." DOI: 10.1093 jnci djh279 Journal of the National Cancer Institute, Vol. 96, No. 20, Oxford University Press 2004, all rights reserved, for example, piracetam price.
Pharmaceutical bioequivalence standards are one of the most important standards regulating the quality of drug products on the world markets today. These standards were debated in the seventies and eighties and codified by the mid-eighties in the United States. The standards that are currently accepted by the US FDA are based on plasma level determinations and include Cmax and AUC. Standards for immediate release products require a single dose study in normal subjects while controlled release products require fasted-fed and steady-state studies. These standards are empirical requiring an in vivo human study to establish bioequivalence. However, there are many oral drug products for which in vivo plasma levels are not influenced by the formulation. A new approach to setting drug bioequivalence standards includes determining the rate determining step for systemic availability, in particular, solubility and permeability. These properties plus a dissolution rate specification may allow for in vitro determination of bioequivalence and ensure in vivo bioequivalence. This approach is being developed by the USA FDA and incorporated into a soon to be released Biopharmaceutics Classification System BCS ; Guidance. For controlled release products the transit through the gastrointestinal tract is more complex. Consequently, classifying controlled release products as to drug release mechanisms, as well as solubility and permeability, is probably essential for establishing in vitro standards for controlled release products. This presentation will review the current status and progress towards establishing a more mechanistic basis for regulating bioequivalence of immediate and controlled release oral drug products based on the BCS!
This emedtv segment lists other common side effects of the drug and also explains which ones should be reported promptly to a healthcare provider, for example, piracetam supplement. 22 ; thus, piracetam must be considered one of the toxicologically safest drugs ever developed. In 1988, Health and Welfare Canada responded to these issues and produced infection control guidelines for long-terni care facilities. In the same year, the US based agency, the Centre for Disease Control tailored its documents on nosocomial infection and surveillance to long-terni care institutions. In 1995, Health Canada updated its document with current procedures and definitional criteria. Research studies in this area can now use standard definitions of Cross and piroxicam.
Sales growth. However, the US operating environment was getting tougher. Managed care, in which plan administrators set cost and reimbursement limits on healthcare services, was also changing market dynamics. In 1990, 63 per cent of prescriptions were paid in cash by patients but by 2001, 73 per cent were paid by managed care plans. As companies' cost for providing drug benefits to employees increased 19 to 20 percent annually, MCOs began to encourage the use of generics through schemes where the consumer paid less if a generic was prescribed and extra for newer drugs. Furthermore, powerful bulk purchasers, such as the Veterans Administration with 6.9 million members, were able to extract prices even lower than those in Canada, so that average US prices paid were actually significantly lower than headline figures in the popular press suggested. Japan has traditionally been the second largest market for pharmaceuticals, with sales of $47 billion in 2002. The Japanese operating environment has historically been very different from that of the US or the EU. This divergence occurred at all levels, from medical practice, healthcare delivery and funding, to regulatory requirements, higher prices, the lack of generics, distribution, and the accepted approach to sales and marketing. Not surprisingly, relatively small domestic companies dominated the market. The Japanese pharmaceutical industry experienced significant environmental turbulence in the 1990s. Following a number of scandals, the system controlling clinical trials and regulatory approvals underwent a major modernisation programme, and many domestic companies were ill equipped to operate to the new standards. The economic recession caused tax revenues to fall, while the cost of treating the world's most rapidly ageing population was rising. This resulted in unprecedented price cuts, changes to healthcare funding and the introduction of stringent price controls. The upshot was very low pharmaceutical market growth of only 1 per cent in 2002. Europe makes up the third part of the Triad, with the top five markets Germany, France, Italy, UK, Spain ; predicted to continue contributing around three quarters of EU sales out to 2007. European markets each have their own unique operating environments but they are generally characterised by strong payer pressures and consequently lower prices than the US or Japan. Combined with slowing economies, these pressures constrained EU market growth to 8 per cent in 2002. Expansion of the EU, however, provided opportunities for growth, especially in Poland and central Europe, but also brought new challenges from generics and low-priced parallel imports. Although growth prospects for emerging markets were considered modest in 2003, their enormous populations and high levels of unmet need offered significant long-term potential. Many had strengthened patent protection and liberalised equity controls. The pharmaceutical markets in Latin America had proved highly volatile, reflecting underlying economic trends. Nevertheless they had large numbers of wealthy consumers who were able to afford branded drugs. Pacific rim countries were becoming more important. Copy products were traditionally a significant issue in these markets, where patent protection was absent or very difficult to police. Pharmaceutical companies focused particularly on China, which had one of the fastest growing pharmaceutical. Mike - I just wish we had an understanding or a correlation of US lab test ranges and what you might be using there in the UK. then I'd feel better about the numbers you are mentioning. For us here. CRP - Reference range: .00 - .50 mg dl. with ideal being as close to zero as possible. Your under 4 number scares me.but then, I don't know how this compares to our ranges. Same thing for the Homocysteine. Cardiologist, Stephen Sinatra says "Generally, "normal" for homocysteine is anything between 5 and 15 micromoles per liter, but epidemiological evidence suggests that optimal levels are less than 8 mm l. Population studies in the Mediterranean Basin France and Spain ; have low mortality from cardiovascular disease, with levels averaging 7 8 mm countries with high mortality rates Finland Scotland, Northern Ireland, Germany ; homocysteine levels average 10 11 mm considers anything above 9 to be dangerous." Sinatra Health Report 3 01 ; Yes - true.you're not that "high" but my doctor is happy when my numbers are 6 or less. This all has to do with methylation and it's work to change the numbers.so forewarned is forearmed. One of holistic doctors speaking on atherosclerosis.this is the danger from the CRP and HCY elevations.says it's high time we start treating the blood to get rid of this problem before it starts becoming an atherosclerotic plaque problem. Prevention is where it's "at." I'm glad to see you are giving blood. That's a powerful step in the right direction, Mike. Good for you. Be well and pletal, for instance, piracetam forum.
Often dramatically. Is usually unanticipated. Should always be considered a MEDICAL EMERGENCY. Results from a Cochrane review Greener et al. 2001 ; , indicate that patients with aphasia associated with stroke, who received piracetam were more likely to experience improvement on any language measure by the end of treatment, compared to those who received placebo OR 0.46 95% CI 0.3-0.7 ; . The authors also noted a nonstatistically significant risk of death associated with piracetam. The efficacy of long-term usage has not yet been examined. Conclusions Regarding Pracetam There is a strong Level 1a ; evidence that piracetam improves aphasia recovery in stroke patients also receiving speech and language therapy over the short-term and premphase!
100 mcg dawk GlaxoSmithKline inhalacyjn Pharmaceuticals S.A. Humulin N 1000 U 10 ml vial Humulin R 1000 U 10 ml vial Imipenem 500 mg vial Imipramine 25 mg tab Infanrix 0.5 ml vial DTPa ; Diphtheria, Tetanus Toxoids & Acellular Pertussis Infanrix-IPV + Hib 0.5 ml vial ; Isomil 40 mg tab Verapamil ; Isormal 20 mg tab Isosorbide-mononitrate ; Keflex 250 mg cap Cephalexin ; Kelfer 500 mg cap Deferiprone ; Keflor 250 mg cap Cefaclor ; Lisamin 250 mg cap Tranexamic Acid ; Lisacef 500 mg cap Cephradine monohydrate ; Leukeran 2 mg tab Chlorambucil ; Leunase 5000 KU vial Asparaginase ; Mebendazole 100 mg tab Mebeverine 100 mg tab Methon 15 mg tab Dextromethorphan ; Mednin 4 mg tab Methylprednisolone ; Meletin 100 mg cap Mexiletine ; Mexitil ; Melzin 25 mg tab Thioridazine ; Methylephedrine 25 mg tab Methylphenidate 10 mg tab Marvelon 21 tab pk Desogestrel & Ethinylestradiol ; Mercilon 21 tab pk Desogestrel & Ethinylestradiol ; Monopril 10 mg tab Fosinopril ; Nootropil 1200 mg tab Pirzcetam ; Nacid 500 mg tab Hydrotalcite ; Nadis 40 mg tab Furosemide ; ONSD oph sol 0.01 %, 10 ml bt Neostigmine ; OSMD eye soln 4 %, 15 ml Sulfamethoxazole ; Sinomin ; Pariet 20 mg tab Rabeprazole ; Parilac 2.5 mg tab Bromocriptin ; Procid 500 mg tab Probenecid ; Procil 50 mg tab Propylthiouracil ; Suprane 240 ml bt Desflurane ; Supren 8 mg 4 ml amp Ondansetron ; Tienam 500 mg vial Imipenem ; Timentin 1600 mg vial Ticarcillin & Clavulanic Potassium ; Ulex Cream 10 g tube Crotamiton ; Ulex susp 25 mg ml, 60 ml bt Cephalexin ; Vancocin 500 mg vial Vancomycin ; Vanconin 5 mg tab Diazepam ; Ventol Liquid 5.34 mg ml, 60 ml bt Theophylline ; Ventolin Nebule 5 mg 2.5 ml nebule Salbutamol ; Vinblastine 10 mg vial Vincristine 1 mg 1 ml vial Vitacal 20 ml amp Calcium Chloride & Dextrose ; Ca: 0.274 mEq ml ; Vitacicol 100 mg 2 ml amp Ascorbic Acid and propranolol.
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The first painter to be called an "Impressionist" is of particular medical interest. Lens opacities had made him legally blind by the time he was in his 60's. During the next ten years his cataracts worsened. His later paintings became increasingly "muddied" with a comparative absence of detail. The bright sunlight caused him difficulty so he changed the hours at which he painted. His ability to distinguish colors diminished and he sometimes used pigments at random. A friend of his, a physician, had become premier of France. He encouraged the painter to paint a group of large panels depicting water lilies. The painter was having so much difficulty seeing that he threatened to quit the project. The painter could see only light with his right eye and had acuity of 20 200 in his left eye. The physician-friend convinced him to have a cataract operation and provera.
Piracetam ebaySchmid has also shown that the incorporation of piracetam a derivative of gamma-amino butyric acid ; in the treatment of ds patients is most advantageous during the first 6-24 months of life. Piracetam mainly affected the map landscape of the time segments following the triplet's last digit and rabeprazole.Fiscal 2002 did not represent a year of full commercial production and included additional costs of plant upscaling including production method adjustment costs. Also included in fiscal 2002 was an amount of approximately $0.8 million of upscaling costs for the Company's development of the fine chemical AD ADD the AD ADD technology was subsequently sold in 2003, see "Divestiture of Technology" under 2003 Highlights, above ; . In the five-month fiscal period ended December 31, 2001 and the year ended July 31, 2001, significant inventory valuation adjustments were included in the Cost of Sales figure relating to start-up and development costs for both the sterols and fine chemicals businesses. Research and development The Company's net R&D expenses for the year ended December 31, 2002, totaled $3.2 million compared with $2.1 million for the five months ended December 31, 2001, and $7.1 million for the year ended July 31, 2001. R&D expenditures for the fiscal year ended July 31, 2001 and the five-month fiscal period ended December 31, 2001 included the operating costs of the Company's laboratory facilities at UBC. Since mid-2002, non-core research, not directly related to the Company's new and more focused R&D project pipeline, has been suspended reducing overall R&D expenditures. In December of 2002 the Company wrote down a net book balance of $1.1 million in laboratory assets including an amount of $0.7 million for leasehold improvements. Also in 2002, laboratory equipment valued at $0.2 million has been transferred to UBC as payment in kind for future research services. $0.16 million of the payment in kind was utilized for research work and expensed in 2003. General and administrative G&A expenses for fiscal year 2002 totaled $4.3 million compared with $2.1 million for the five-month fiscal period ended December 2001 and $7.0 million in fiscal year ended July 31, 2001. In 2002 G&A showed an overall reduction from the five-month fiscal period ended December 31, 2001 and fiscal 2001 primarily attributable to downsized operations including the reduction in staff levels, resulting in lower personnel costs and related expenses. By type of costs incurred, G&A for 2002 consists of professional services - $1.3 million 5-month fiscal period ended December 31, 2001 - $0.5 million; fiscal year ended December 31, 2001 - $1.6 million ; , salaries and benefits - $0.7 million 5-month fiscal period ended December 31, 2001 - $0.8 million; fiscal year ended December 31, 2001 - $2.9 million travel - $0.2 million 5-month fiscal period ended December 31, 2001 - $0.1 million; fiscal year ended December 31, 2001 - $0.3 million occupancy costs - $0.4 million 5-month fiscal period ended December 31, 2001 - $0.2 million; fiscal year ended December 31, 2001 - $0.7 million and operations - $1.7 million 5-month fiscal period ended December 31, 2001 - $0.5 million; fiscal year ended December 31, 2001 - $1.5 million ; . In 2002, cost cutting measures were implemented and some wages were deferred and repaid in 2003. Wages in 2002 included a net reduction of tenure allowance liability of approximately $0.2 million. An additional component of G&A is an amount for stock based compensation expense recorded in 2002 of $0.02 million 5-month fiscal period ended December 31, 2001 and fiscal year ended December 31, 2001 - Nil ; . Depreciation and amortization "Amortization" ; Amortization expense for 2002 relates to the amortization of equipment and intangible assets acquired upon the formation of the Phyto-Source joint venture. For the five months ended December 31, 2001 Amortization relates mainly to the depreciation of laboratory equipment and leasehold improvements for the laboratory space at UBC. In Fiscal 2001, Amortization relates largely to the depreciation of assets at the Company's Amqui pilot plant facility. For the year ended December 31.
| Piracetam 400What countries do you piracetaj ship to and ramipril. 1. Take your pill s ; as directed by your doctor, pharmacist and or diabetes team. You may be taking more than one kind of pill, so it's a good idea to take your pill s ; at the same time everyday. A weekly schedule is included in this package that you can use to write down which pills you take throughout the day. 2. Store your pill s ; in the original container that they come in. This bottle will have the directions and other important information for you and your family. Many pills are light and heat sensitive, so keep them in the bottle that the pharmacy gives you. 3. Do not self adjust your medication s ; unless your doctor has instructed you to. If your doctor has told you to take your pill s ; 3 times a day with meals, don't double up, or decrease without speaking to your doctor and or diabetes team. 4. You may be able to see your blood sugar drop within one day after starting your medication s however it may take 2 to 3 months to see the full effects on your blood sugar level A1C ; . It is important to keep taking your pill s ; as directed. 5. If you notice any side effects, call your pharmacist before you stop taking your medication s ; . A contact sheet is included in this package where you can write down your pharmacist's phone number. 6. Along with taking your medication s ; properly, it is important to have a healthy diet and an active lifestyle. 7. It is also important to take your other medications that your doctor has ordered for you to prevent future heart problems like controlling your blood pressure and cholesterol level.Piracetam nootropil nootropics doses |
Keywords Composting toilets, ecological sanitation, El Salvador, latrines, solar toilets, hygiene, bacteria, helminths, viruses, protozoa, die-off Abstract This one-year study took repeated measures of the physical moisture content, temperature ; , chemical pH ; and microbiological faecal coliforms, Clostridium perfringens, coliphage, Trichuris, hookworm and Ascaris ; properties of biosolids from 156 ecosan toile ts in El Salvador with different designs, use and maintenance patterns. In double -vault urine diverting toilets, pH and long storage time were the primary factors that contributed to microbial inactivation. In solar toilets with black metal covers ; , hi gh peak temperature, pH and storage time were the critical factors affecting microbial inactivation. This study demonstrates that improvements in toilet design larger, partitioned vaults and good solar exposure ; , longer storage time and consistent use of high pH additives will result in an endproduct that meets USEPA standards for Class A biosolids that are safe for agricultural use. Introduction El Salvador is the most densely populated country in Central America, and ecological sanitation ecosan ; has been promoted by the Ministry of Health, UNICEF and several non-government organizations. Double-vault urine diverting toilets DVUD ; and single -vault solar toilets are intended to store faecal waste under conditions that promote desiccation and inactivati on of faecal pathogens. The treated biosolids are then removed and disposed of or may be used for agricultural purposes. We previously reported on a baseline survey of over 400 households with DVUD or solar toilets in six rural and one urban community in El Salvador where we observed a wide range in the physical characteristics and microbial quality of the stored biosolids Moe et al., 2001 ; . We now report on a longitudinal study of 156 DVUD and solar latrines in seven rural communities in El Salvador fro m November 2000 to March 2002. Methods Field Methods: We conducted a survey of 118 households with DVUD toilets and 38 households with solar toilets in seven rural communities in El Salvador. The toilets had been in operation for an average of 5.4 years, with a range of 1 to years. The head of each and rimonabant.
Although the labeler of these products provided a DESI Code 2 safe and effective ; for each NDC, the FDA has determined that these drugs are less-than-effective, or a DESI Code 5. North Carolina has made this change to the drug file.
Feb 10, 2006 ; . Retrieved July 7, 2006. from : fda.gov cder drug advisory benzocaine . Benzocaine sprays are used in medical practice for locally numbing mucous membranes of the mouth and throat for minor surgical procedures or when a tube must be inserted into the stomach or airways. Their use is known to be occasionally associated with methemoglobinemia. However, cases of methemoglobinemia have also resulted from medication errors due to incorrect use of benzocaine sprays e.g., longer duration or more frequent sprays than recommended. ; On February 8, 2006, the Veterans Health Administration VA ; announced the decision to stop using benzocaine sprays for these purposes. The FDA is aware of the reported adverse events and is reviewing all available safety data, but at this time is not planning action to remove the drugs from the market. At present, the FDA suggests considering the following points about the use of benzocaine sprays in procedures requiring that a tube be inserted in the larynx or pharynx down the throat, windpipe, or gullet ; or in minor surgical procedures performed in these locations. Considerations Benzocaine sprays used in the mouth and throat can result in potentially dangerous levels of methemoglobinemia. Patients who have breathing problems such as asthma, bronchitis, or emphysema, patients with heart disease, and patients who smoke are at greater risk for complications related to methemoglobinemia and may be candidates for other forms of therapy. Patients who may have greater tendency for elevated levels of methemoglobinemia, such as all children less than 4 months of age and older patients with certain in-born defects such as glucose-6-phosphodiesterase G-6-PD ; deficiency, hemoglobin-M disease, NADH-methemoglobin reductase diaphorase 1 ; deficiency, and pyruvate-kinase deficiency ; may benefit from products with different active ingredients such as lidocaine. Patients who receive benzocaine sprays should be given the minimum amount needed, to reduce the risks associated with methemoglobinemia. Patients who receive benzocaine sprays should be carefully observed for signs of methemoglobinemia including pale, gray or blue colored skin, headache, lightheadedness, shortness of breath, anxiety, fatigue and tachycardia rapid heart rate ; . Methemoglobinemia makes a standard device used to assess the amount of oxygen bound to hemoglobin 2-wavelength pulse oximetry ; unreliable. If blood is drawn to check for the condition, a co-oximeter is needed to reliably detect methemoglobinemia. A characteristic color of the blood chocolate-brown rather than blood-red ; can be a danger sign, but these changes are a late sign of the condition. Patients suspected of having high levels of methemoglobinemia should be promptly treated. Methemoglobinemia is a condition where too much of the hemoglobin in red blood cells becomes unable to bind and carry oxygen. While there is treatment available, until the condition is reversed, oxygen is not effectively delivered throughout the patient's body. Patients with methemoglobinemia can suffer effects ranging from headache to cyanosis turning blue due to lack of oxygen ; that can be life-threatening in the most severe cases. Patients with underlying breathing problems, such as asthma or emphysema, patients with heart disease, and those who smoke may be more susceptible to the problems from methemoglobinemia and may suffer from bad effects from this condition at lower levels of methemoglobin than healthy individuals. Similarly, some patients may lack or have reduced level of enzymes that help reverse the methemoglobinemia, and they are also more susceptible to risks associated with benzocaine sprays.
Research has shown these provisions to be effective for this group of users. Gossop et al., 2003; US Department of Health and Human Services, 1999 ; . Regarding specific groups: Controversial evidence suggests that the use of stimulant therapy for cocaine co-abusing problem users with Attention Deficit Hyperactive Disorder might reduce cocaine coconsumption Schubiner et al., 2002; Levin et al., 1998 ; . Clients in substitution treatment for opioid dependence MMT, BMT ; might benefit from an enhancement of the dose of the respective substitute Maxwell & Shinderman, 2002; Strain et al., 1999 ; , or from the more frequent granting of take-home doses for those who are sufficiently stabilised Rhoades et al., 1998 ; . Therefore, adherence to the relevent European guidelines should be encouraged. Moreover, there are claims that the provision of heroin to severely dependent heroin and cocaine users might be useful to reduce co-consumption as well Blttler et al., 2002 ; . Improving access to main-stream medical services is of great importance given the welldocumented "under-consumption" of psychiatric services by this group. Finally, the involvement of private practitioners in the treatment of problem users should be achieved by better training and more adequately calculated fees. Recreational users Intervention methods for recreational users generally consists of providing them with accurate information about the long and short-term effects of party-drugs and combined usage. There is very little scientific evidence with on the effectiveness of prevention strategies for this group. Some groups have started on-site pill testing to inform users about the dangerous and unexpected contents found in pills. This practice remains controversial due to its potential for conveying the wrong safety messages to consumers Koesters et al., 2002; Kriener et al., 2001.
Studies were identified by searching medline, embase excerpta medica, international pharmaceutical abstracts, biological abstracts, lilacs, psyclit, the cochrane library, and the trial register of the cochrane depression, anxiety, and neurosis group january 1972 to december 1998 hand searching major mental health and general medicine journals; scanning the reference lists of identified articles; checking scisearch; and making personal contacts, for instance, pirace6am side effects.
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1. Adams HP, Brott TG, Crowell RM, Furlan AJ, Gomez CR, Grotta J, Helgason CM, Marler JR, Woolson RF, Zivin JA, et al.: Guidelines for the management of patients with acute ischemic stroke. A statement for healthcare professionals from a special writing group of the Stroke Council, American Heart Association. Stroke 25: 1901-1914., 1994 Adams HP, Brott TG, Furlan AJ, Gomez CR, Grotta J, Helgason CM, Kwiatkowski T, Lyden PD, Marler JR, Torner J, Feinberg W, Mayberg M, Thies W: Guidelines for Thrombolytic Therapy for Acute Stroke: a Supplement to the Guidelines for the Management of Patients with Acute Ischemic Stroke. A statement for healthcare professionals from a Special Writing Group of the Stroke Council, American Heart Association. Stroke 27: 1711-1718., 1996 Boysen G: Cerebral hemodynamics in carotid surgery. Acta Neurologica Scandinavica 49 suppl 52 ; : 3-86, 1973 4. Broderick J, Brott T, Barsan W, Haley EC, Levy D, Marler J, Sheppard G, Blum C: Blood pressure during the first minutes of focal cerebral ischemia. Ann Emerg Med 22: 1438-1443., 1993 Brott T, Lu M, Kothari R, Fagan SC, Frankel M, Grotta JC, Broderick J, Kwiatkowski T, Lewandowski C, Haley EC, Marler JR, Tilley BC: Hypertension and its treatment in the NINDS rt-PA Stroke Trial. Stroke 29: 1504-1509., 1998 Chamorro A, Vila N, Ascaso C, Elices E, Schonewille W, Blanc R: Blood pressure and functional recovery in acute ischemic stroke. Stroke 29: 1850-1853., 1998 Collins R, Peto R, MacMahon S, et al: Blood pressure, stroke, and coronary heart disease. Part 2. Short term reductions in blood pressure: overview of randomized drug trials in their epidemiological context. Lancet 335: 827-838, 1990 Dirnagl U, Pulsinelli W: Autoregulation of cerebral blood flow in experimental focal brain ischemia. J Cereb Blood Flow Metab 10: 327-336, 1990 Goldstein LB: Should antihypertensive therapies be given to patients with acute ischaenic stroke? Drug Saftey 22: 13-18, 2000 Grubb RL Jr, Raichle ME, Phelps ME, Ratcheson RA: Effects of increased intracranial pressure on cerebral blood volume, blood flow, and oxygen utilization in monkey. J Neurosurg 43: 385-398, 1975 Heistad DD, Kontos HE eds ; : Cerebral Circulation. Bethesda: American Physiological Society, 1983, Vol 3 12. Kety SS, King BD, Horvath SM, Jeffers WA, Hafkenschiel JH: The effects of an acute reduction in blood pressure by means of differential spinal sympathetic block on the cerebral circulation of hypertensive patients. J Clin Invest 29: 402-407, 1950 Lennox WG, Gibbs FA, Gibbs EL: Relationship of unconsciousness to cerebral blood flow and to anoxemia. Arch Neurol Psych 34: 1001-1013, 1935 MacKenzie ET, Farrar JK, Fitch W, Graham DI, Gregory PC, Harper AM: Effects of hemorrhagic hypotension on the cerebral circulation: I.cerebral blood flow and pial arteriolar caliber. Stroke 10: 711-718, 1979.
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